Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Carola Tapia-Monsalves"'
Autor:
Rodrigo A. Quintanilla, Carola Tapia-Monsalves, Erick H. Vergara, María José Pérez, Alejandra Aranguiz
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 14 (2020)
Mitochondria are highly specialized organelles essential for the synapse, and their impairment contributes to the neurodegeneration in Alzheimer’s disease (AD). Previously, we studied the role of caspase-3–cleaved tau in mitochondrial dysfunction
Externí odkaz:
https://doaj.org/article/d967ec8aff044517a8f7d4f1338203dc
Autor:
Felipe Bodaleo, Carola Tapia-Monsalves, Christian Cea-Del Rio, Christian Gonzalez-Billault, Alexia Nunez-Parra
Publikováno v:
Frontiers in Molecular Neuroscience, Vol 12 (2019)
Fragile X Syndrome (FXS) is the most common inherited form of intellectual disability. It is produced by mutation of the Fmr1 gene that encodes for the Fragile Mental Retardation Protein (FMRP), an important RNA-binding protein that regulates the exp
Externí odkaz:
https://doaj.org/article/96578da1c6f04a53ac793ace28952e48
Autor:
Carola Tapia-Monsalves, Margrethe A. Olesen, Francisca Villavicencio-Tejo, Rodrigo A. Quintanilla
Publikováno v:
Molecular and Cellular Neuroscience. 125:103861
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f1e54d06fae8368b05dcf8207e0db95f
https://doi.org/10.1016/j.mcn.2023.103861
https://doi.org/10.1016/j.mcn.2023.103861
Publikováno v:
Current Neuropharmacology
Accumulative evidence has shown that mitochondrial dysfunction plays a pivotal role in the pathogenesis of Alzheimer's disease (AD). Mitochondrial impairment actively contributes to the synaptic and cognitive failure that characterizes AD. The presen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8b7b2541c7b0a4fd952629d2a4cd12e2
https://doi.org/10.2174/1570159x18666200525020259
https://doi.org/10.2174/1570159x18666200525020259
Autor:
Carola Tapia-Monsalves, María José Pérez, Alejandra Aranguiz, Rodrigo A. Quintanilla, Gloria Mendez
Publikováno v:
Neurotoxicity research. 38(2)
Excessive alcohol intake affects hippocampal function and neuronal communication through oxidative stress and mitochondrial impairment. Previous studies have suggested that the melanocortin system (MCS) plays an essential role in alcohol consumption
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::349d3e91d3a74769831285047d4e122c
https://pubmed.ncbi.nlm.nih.gov/32378055
https://pubmed.ncbi.nlm.nih.gov/32378055
Autor:
Francisco Canelo, María José Pérez, Rocío Loyola, Carola Tapia-Monsalves, Alejandra Aranguiz, Cesar Osorio-Fuentealba, Rodrigo A. Quintanilla
Publikováno v:
Neuropharmacology. 171:108100
Acute ethanol treatment induces neurodegeneration in cultured neurons and can lead to brain damage in animal models. Neuronal cells exposed to ethanol showed an increase in reactive oxygen species (ROS), oxidative damage and mitochondrial impairment
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0f4f76ad8550e028eb9187aec71853f7
https://doi.org/10.1016/j.neuropharm.2020.108100
https://doi.org/10.1016/j.neuropharm.2020.108100