Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Carmen Ochmann"'
Autor:
Melanie Ziegler, Bernhard Nieswandt, Götz Münch, Ildiko Konrad, Carmen Ochmann, Klaus T. Preissner, Tobias Jürgens, Margitta Elvers, Steffen Massberg, Peter Seizer, Tanja Schönberger, Meinrad Gawaz, Oliver Borst, Harald F. Langer, Martin Ungerer
Publikováno v:
American Journal of Physiology-Cell Physiology. 303:C757-C766
Platelets play a critical role in the pathophysiology of reperfusion, sepsis, and cardiovascular diseases. In a multiple step process, they adhere to activated endothelium and release proinflammatory cytokines thereby promoting the inflammatory proce
Autor:
Oliver Borst, Harald F. Langer, Rebecca Schleicher, Konstantinos Stellos, Karin Klingel, Martina Sauter, Peter Seizer, Eva-Maria Schmidt, Carmen Ochmann, Andreas E. May, Dirk Westermann, Boris Bigalke, Reinhard Kandolf, Meinrad Gawaz, Tanja Schönberger
Publikováno v:
Journal of Molecular and Cellular Cardiology. 53:6-14
Extracellular cyclophilin A (CyPA) and its receptor Extracellular Matrix Metalloproteinase Inducer (EMMPRIN, CD147) modulate inflammatory processes beyond metalloproteinase (MMP) activity. Recently, we have shown that CyPA and CD147 are upregulated i
Autor:
Romana A. Nowak, Oliver Borst, Peter Seizer, Florian Lang, Karin Klingel, Melanie Rose, Andreas E. May, Stefan Engelhardt, Carmen Ochmann, Sebastian Zach, Tanja Schönberger, H. Robson MacDonald, Meinrad Gawaz, Reinhard Kandolf
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology, vol. 31, no. 6, pp. 1377-1386
Objective— Inflammation and proteolysis crucially contribute to myocardial ischemia and reperfusion injury. The extracellular matrix metalloproteinase inducer EMMPRIN (CD147) and its ligand cyclophilin A (CyPA) may be involved in both processes. Th
Autor:
Carmen Ochmann, Christoph Jacoby, Konstantinos Stellos, Ulrich Flögel, Florian Lang, Meinrad Gawaz, Tanja Schönberger, Oliver Borst, Peter Seizer
Publikováno v:
Cellular Physiology and Biochemistry. 28:1-12
Myocardial ischemia und subsequent reperfusion is followed by a complex sequence of pathophysiological responses involving inflammatory cell infiltration and cytokine release as well as postinfarction wound healing and myocardial tissue remodeling. W
The recombinant bifunctional protein αCD133–GPVI promotes repair of the infarcted myocardium in mice
Autor:
Götz Münch, Melanie Ziegler, B. Schmid-Horch, Yvonne Baumer, Burkhard Schlosshauer, M. Gawaz, Heidrun Degen, A. Perk, Carmen Ochmann, Christoph Leder, Martin Ungerer, Margitta Elvers, Tanja Schönberger
Publikováno v:
Baumer, Y, Leder, C, Ziegler, M, Schönberger, T, Ochmann, C, Perk, A, Degen, H, Schmid-Horch, B, Elvers, M, Münch, G, Ungerer, M, Schlosshauer, B & Gawaz, M 2012, ' The recombinant bifunctional protein αCD133–GPVI promotes repair of the infarcted myocardium in mice ', Journal of Thrombosis and Haemostasis, vol. 10, no. 6, pp. 1152-1164 . https://doi.org/10.1111/j.1538-7836.2012.04710.x
Baumer, Y, Leder, C, Ziegler, M, Schönberger, T, Ochmann, C, Perk, A, Degen, H, Schmid-Horch, B, Elvers, M, Münch, G, Ungerer, M, Schlosshauer, B & Gawaz, M 2012, ' The recombinant bifunctional protein αCD133–GPVI promotes repair of the infarcted myocardium in mice ' Journal of Thrombosis and Haemostasis, vol 10, no. 6, pp. 1152-1164 . DOI: 10.1111/j.1538-7836.2012.04710.x
Baumer, Y, Leder, C, Ziegler, M, Schönberger, T, Ochmann, C, Perk, A, Degen, H, Schmid-Horch, B, Elvers, M, Münch, G, Ungerer, M, Schlosshauer, B & Gawaz, M 2012, ' The recombinant bifunctional protein αCD133–GPVI promotes repair of the infarcted myocardium in mice ' Journal of Thrombosis and Haemostasis, vol 10, no. 6, pp. 1152-1164 . DOI: 10.1111/j.1538-7836.2012.04710.x
Background: Bone-marrow-derived progenitor cells are important in myocardial repair mechanisms following prolonged ischemia. Cell-based therapy of diseased myocardium is limited by a low level of tissue engraftment.Objectives: The aim of this study w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c8c136e0c87845979298df1335862213
http://www.scopus.com/inward/record.url?scp=84861657646&partnerID=8YFLogxK
http://www.scopus.com/inward/record.url?scp=84861657646&partnerID=8YFLogxK
Autor:
Oleg Lunov, Christoph Leder, Tobias Geisler, Margitta Elvers, Tanja Schönberger, Burkhard Schlosshauer, Stefan Engelhardt, Thomas Simmet, Yvonne Baumer, Tobias Jürgens, Meinrad Gawaz, Melanie Ziegler, Carmen Ochmann
Publikováno v:
Ziegler, M, Elvers, M, Baumer, Y, Leder, C, Ochmann, C, Schönberger, T, Jürgens, T, Geisler, T, Schlosshauer, B, Lunov, O, Engelhardt, S, Simmet, T & Gawaz, M 2012, ' The bispecific SDF1-GPVI fusion protein preserves myocardial function after transient ischemia in mice. ', Circulation, vol. 125, no. 5, pp. 685-696 . https://doi.org/10.1161/CIRCULATIONAHA.111.070508
Ziegler, M, Elvers, M, Baumer, Y, Leder, C, Ochmann, C, Schönberger, T, Jürgens, T, Geisler, T, Schlosshauer, B, Lunov, O, Engelhardt, S, Simmet, T & Gawaz, M 2012, ' The bispecific SDF1-GPVI fusion protein preserves myocardial function after transient ischemia in mice. ' Circulation, vol 125, no. 5, pp. 685-696 . DOI: 10.1161/CIRCULATIONAHA.111.070508
Ziegler, M, Elvers, M, Baumer, Y, Leder, C, Ochmann, C, Schönberger, T, Jürgens, T, Geisler, T, Schlosshauer, B, Lunov, O, Engelhardt, S, Simmet, T & Gawaz, M 2012, ' The bispecific SDF1-GPVI fusion protein preserves myocardial function after transient ischemia in mice. ' Circulation, vol 125, no. 5, pp. 685-696 . DOI: 10.1161/CIRCULATIONAHA.111.070508
BACKGROUND: CXCR4-positive bone marrow cells (BMCs) are critically involved in cardiac repair mechanisms contributing to preserved cardiac function. Stromal cell-derived factor-1 (SDF-1) is the most prominent BMC homing factor known to augment BMC en
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fd3c51405b155623c4d81d6502143e81
http://fox.leuphana.de/portal/de/publications/the-bispecific-sdf1gpvi-fusion-protein-preserves-myocardial-function-after-transient-ischemia-in-mice(08bb8040-0ac2-4b20-9a1a-5792ddce6dca).html
http://fox.leuphana.de/portal/de/publications/the-bispecific-sdf1gpvi-fusion-protein-preserves-myocardial-function-after-transient-ischemia-in-mice(08bb8040-0ac2-4b20-9a1a-5792ddce6dca).html