Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Carlyn K. Rosenberg"'
Autor:
Kerstin Henkel, Harald Hampel, Kurt A. Jellinger, Daniela C Glatz, Yesheng Tang, Carlyn K. Rosenberg, Peter Riederer, Christine M. Hulette, Athena Andreadis, Frank Faltraco, Hans-J Möller, Annette M. Hartmann, Dan Rujescu, Frank J. Berendt, Stefan Stamm
Publikováno v:
Journal of Neurochemistry. 96:635-644
Pathological inclusions containing fibrillar aggregates of hyperphosphorylated tau protein are a characteristic feature in tauopathies, which include Alzheimer's disease (AD). Tau is a microtubule-associated protein whose transcript undergoes alterna
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8b07f6fcdd9b6345b0520f16aa18b264
https://doi.org/10.1111/j.1471-4159.2005.03552.x
https://doi.org/10.1111/j.1471-4159.2005.03552.x
Autor:
Carlyn K. Rosenberg, Hans A. Kretzschmar, Michaela Neumann, Stefan J. Teipel, Rusti Oshida, Hans-Jürgen Möller, Dieter Pongratz, Josef Unger, Frank Padberg, Michael Ewers, Petra Fischer, Andreas Haslinger, Christine M. Hulette, Harald Hampel, Michael Scheloske
Publikováno v:
European Archives of Psychiatry and Clinical Neuroscience. 255:269-278
Involvement of the interleukin-6 receptor complex (IL-6RC) in neuroregulatory and immunological processes of the brain and particularly in Alzheimer's disease (AD) has been hypothesized. The functionally active IL-6RC consists of the cytokine IL-6, w
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bb878e6180dec465da051e790eccc5d2
https://doi.org/10.1007/s00406-004-0558-2
https://doi.org/10.1007/s00406-004-0558-2
Autor:
Carlyn K. Rosenberg, John R. Gilbert, Christine M. Hulette, Ann M. Saunders, Margaret A. Pericak-Vance, P. C. Gaskell
Publikováno v:
Acta Neuropathologica. 100:145-152
Mutations in the amyloid precursor protein (APP) gene cause one form of early onset familial Alzheimer's disease (AD). One such family has been studied genetically and neuropathologically and represents the basis of the present report. Four siblings
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d00ecb5fef38c6582f15a129289c01de
https://doi.org/10.1007/s004019900155
https://doi.org/10.1007/s004019900155
Autor:
Charleen T. Chu, James L. Caruso, John F. Ervin, Christine M. Hulette, Thomas J. Cummings, Carlyn K. Rosenberg
Publikováno v:
Modern Pathology. 13:420-426
Alzheimer’s disease is the most common cause of dementia. It is associated with genetic risk factors and at least three autosomal dominant mutations. Community pathologists are frequently asked by families to evaluate autopsy material for Alzheimer
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e8846eb063ecd085f230eb014f17b47f
https://doi.org/10.1038/modpathol.3880072
https://doi.org/10.1038/modpathol.3880072
Publikováno v:
Pediatric Nephrology. 13:223-229
Matrix metalloproteinases (MMPs) are a family of at least 14 zinc-dependent proteinases that have been implicated in matrix turnover under both normal and pathological conditions. Previous studies have shown that several MMPs are produced in various
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f50ced9e93a8cbb1323a4918a1a9f361
https://doi.org/10.1007/s004670050597
https://doi.org/10.1007/s004670050597
Autor:
Lauren M. McIntyre, Christine M. Hulette, Carlyn K. Rosenberg, Thomas J. Cummings, Corinne Widico, Ann M. Saunders
Publikováno v:
Acta neuropathologica. 102(6)
To investigate similarities and differences between Alzheimer's disease (AD) and dementia with Lewy bodies (DLB), we undertook a demographic analysis of 277 patients from the Kathleen Price Bryan Brain Bank with an antemortem diagnosis of probable AD
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::04ae7b575b83399341b70967c34c27c2
https://pubmed.ncbi.nlm.nih.gov/11761723
https://pubmed.ncbi.nlm.nih.gov/11761723
Publikováno v:
Archives of neurology. 57(6)
Nerve growth factor (NGF), brain-derived neurotrophic factor (BDNF), neurotrophin 3 (NT-3), and neurotrophin 4/5 (NT-4/5) are members of the neurotrophin gene family that support the survival of specific neuronal populations, including those that are
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d0d255f41f1acef8ad5dacf0c9340902
https://pubmed.ncbi.nlm.nih.gov/10867782
https://pubmed.ncbi.nlm.nih.gov/10867782
Autor:
Uwe Otten, Christoph Hock, Franz Müller-Spahn, Klaus Heese, Carlyn K. Rosenberg, Christine M. Hulette
Publikováno v:
Neuroscience letters. 241(2-3)
The cholinergic neurons of the basal forebrain system are sensitive to nerve growth factor (NGF), a member of the neurotrophin gene family. Since the cholinergic system is affected early in the course of Alzheimer's disease (AD), it was hypothesized
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b2bce6da13ccbe20410ae858fc5bfe7c
https://pubmed.ncbi.nlm.nih.gov/9507943
https://pubmed.ncbi.nlm.nih.gov/9507943
Autor:
Carlyn K. Rosenberg, Delila Serra, John L. Fowlkes, Kathryn M. Thrailkill, Carlos George-Nascimento
Publikováno v:
Endocrinology. 138(6)
MC3T3-E1 murine osteoblasts produce insulin-like growth factor (IGF)-binding protein-4 (IGFBP-4)-degrading proteinase activity, which is inhibited by IGFBP-3 and a highly basic, C-terminal domain of IGFBP-3. Of all the other five IGFBPs, IGFBP-5 and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3dd2d3d0f3c7933b4512854a0228e97f
https://pubmed.ncbi.nlm.nih.gov/9165012
https://pubmed.ncbi.nlm.nih.gov/9165012
Publikováno v:
The Journal of biological chemistry. 270(46)
Previous studies have shown that insulin-like growth factor (IGF)-binding protein-4 (IGFBP-4) is degraded only in the presence of exogenous IGFs; however, we found that cation-dependent proteinase activity present in conditioned medium of MC3T3-E1 os
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::870d6c9bbb61955eacd1bf20322931f2
https://pubmed.ncbi.nlm.nih.gov/7499205
https://pubmed.ncbi.nlm.nih.gov/7499205