Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Carla Macheroni"'
Autor:
Deborah Simão Souza, Carla Macheroni, Gustavo José Silva Pereira, Carolina Meloni Vicente, Catarina Segreti Porto
Publikováno v:
Frontiers in Endocrinology, Vol 14 (2023)
Prostate cancer remains the most prevalent cancer among men worldwide. This cancer is hormone-dependent; therefore, androgen, estrogen, and their receptors play an important role in development and progression of this disease, and in emergence of the
Externí odkaz:
https://doaj.org/article/18d09e88ca574b9f86272802040d4721
Publikováno v:
Heliyon, Vol 6, Iss 11, Pp e05363- (2020)
The aim of the study was to investigate the effects of estrogen receptors (ESR1 and ESR2) on the expression of the proteins involved with proliferation (CCND1) and differentiation (CDKN1B and CTNNB) of Sertoli cells from rat in different stages of de
Externí odkaz:
https://doaj.org/article/e72458af729742cfae63cd63631103e0
Autor:
Deborah Souza, Carla Macheroni, Carolina Vicente, Renan Cavalheiro, Vanessa Campo, Catarina Porto
Publikováno v:
Oncology Reports. 49
Autor:
Carla Macheroni, Thaís Fabiana Gameiro Lucas, Deborah Simão Souza, Carolina Meloni Vicente, Gustavo José da Silva Pereira, Itabajara da Silva Vaz Junior, Maria Aparecida Juliano, Catarina Segreti Porto
Publikováno v:
Molecular and cellular endocrinology. 554
The aims of the present study were to investigate the expression of the classic estrogen receptors ESR1 and ESR2, the splicing variant ESR1-36 and GPER in human testicular embryonal carcinoma NT2/D1 cells, and the effects of the activation of the ESR
Publikováno v:
Heliyon, Vol 6, Iss 11, Pp e05363-(2020)
Heliyon
Heliyon
The aim of the study was to investigate the effects of estrogen receptors (ESR1 and ESR2) on the expression of the proteins involved with proliferation (CCND1) and differentiation (CDKN1B and CTNNB) of Sertoli cells from rat in different stages of de
Autor:
Maria Fatima M. Lazari, Catarina S. Porto, Aline R. Nascimento, Thaís F.G. Lucas, Carla Macheroni
Publikováno v:
Reproduction (Cambridge, England). 152(6)
Follicle-stimulating hormone (FSH) stimulates the proliferation of immature Sertoli cells through the activation of PI3K/AKT/mTORC1 and MEK/ERK1/2 pathways. Mature Sertoli cells stop proliferating and respond to FSH by stimulating cAMP production. To