Zobrazeno 1 - 10
of 40
pro vyhledávání: '"Carl Wibom"'
Autor:
Adam Rosenbaum, Anna M. Dahlin, Ulrika Andersson, Benny Björkblom, Wendy Yi-Ying Wu, Håkan Hedman, Carl Wibom, Beatrice Melin
Publikováno v:
Scientific Reports, Vol 13, Iss 1, Pp 1-9 (2023)
Abstract Genome-wide association studies (GWAS) have contributed to our understanding of glioma susceptibility. To date, 25 risk loci for development of any of the glioma subtypes are known. However, GWAS studies reveal little about the molecular pro
Externí odkaz:
https://doaj.org/article/fd0f3a703d9e4e7c89cbe908bd914f26
Autor:
Wendy Yi‐Ying Wu, Florentin Späth, Carl Wibom, Benny Björkblom, Anna M. Dahlin, Beatrice Melin
Publikováno v:
Cancer Medicine, Vol 11, Iss 4, Pp 1016-1025 (2022)
Abstract No strong aetiological factors have been established for glioma aside from genetic mutations and variants, ionising radiation and an inverse relationship with asthmas and allergies. Our aim was to investigate the association between pre‐di
Externí odkaz:
https://doaj.org/article/96b7b1bce85a4b789d8e6dbb8f515c86
Autor:
Florentin Späth, Carl Wibom, Esmeralda J. M. Krop, Antonio Izarra Santamaria, Ann-Sofie Johansson, Ingvar A. Bergdahl, Johan Hultdin, Roel Vermeulen, Beatrice Melin
Publikováno v:
Haematologica, Vol 104, Iss 12 (2019)
Biomarkers reliably predicting progression to multiple myeloma (MM) are lacking. Myeloma risk has been associated with low blood levels of monocyte chemotactic protein-3 (MCP-3), macrophage inflammatory protein-1 alpha (MIP-1α), vascular endothelial
Externí odkaz:
https://doaj.org/article/d49abad318df4d75bc4e3d1f1367fbf8
Autor:
Matilda Rentoft, Daniel Svensson, Andreas Sjödin, Pall I Olason, Olle Sjöström, Carin Nylander, Pia Osterman, Rickard Sjögren, Sergiu Netotea, Carl Wibom, Kristina Cederquist, Andrei Chabes, Johan Trygg, Beatrice S Melin, Erik Johansson
Publikováno v:
PLoS ONE, Vol 14, Iss 3, p e0213350 (2019)
Whole-genome sequencing is a promising approach for human autosomal dominant disease studies. However, the vast number of genetic variants observed by this method constitutes a challenge when trying to identify the causal variants. This is often hand
Externí odkaz:
https://doaj.org/article/9b8a06b2b5274ef9a69fa9d3f6ad38d7
Publikováno v:
PLoS ONE, Vol 11, Iss 10, p e0163067 (2016)
Genome-wide association studies and candidate gene studies have identified several genetic variants that increase glioma risk. The majority of these variants are non-coding and the mechanisms behind the increased risk in carriers are not known. In th
Externí odkaz:
https://doaj.org/article/ff0464059fa44dd8824393bd64054c3d
Autor:
Carl Wibom, Soma Ghasimi, Peter Van Loo, Thomas Brännström, Johan Trygg, Ching Lau, Roger Henriksson, Tommy Bergenheim, Ulrika Andersson, Patrik Rydén, Beatrice Melin
Publikováno v:
PLoS ONE, Vol 7, Iss 12, p e47929 (2012)
A number of gene variants have been associated with an increased risk of developing glioma. We hypothesized that the reported risk variants may be associated with tumor genomic instability. To explore potential correlations between germline risk vari
Externí odkaz:
https://doaj.org/article/9145a39e940447aaa7491cf9ba677e7c
Autor:
Beatrice Melin, Roel Vermeulen, Ingvar A. Bergdahl, Ann-Sofie Johansson, Esmeralda J.M. Krop, Carl Wibom, Florentin Späth
Case-only analysis for linear mixed effect modeling.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::09470a5c02c82e79adab3a34a2e0f943
https://doi.org/10.1158/0008-5472.22416192.v1
https://doi.org/10.1158/0008-5472.22416192.v1
Autor:
Beatrice Melin, Roel Vermeulen, Ingvar A. Bergdahl, Ann-Sofie Johansson, Esmeralda J.M. Krop, Carl Wibom, Florentin Späth
The B-cell activation markers CXCL13, sCD23, sCD27, and sCD30 are associated with future lymphoma risk. However, a lack of information about the individual dynamics of marker–disease association hampers interpretation. In this study, we identified
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1dbeae3de8a912668e4db7edb2ea7e2d
https://doi.org/10.1158/0008-5472.c.6509484.v1
https://doi.org/10.1158/0008-5472.c.6509484.v1
Autor:
Beatrice Melin, Ulrika Andersson, Tom Børge Johannesen, Preetha Rajaraman, Eivind Hovig, Hilde Langseth, Anna M. Dahlin, Florentin Späth, Carl Wibom
Supplementary Tables 1-7 Supplementary Table 1. PCR primers and conditions Supplementary Table 2. List of included diagnoses in each histological subgroup Supplementary Table 3. Associations between published genetic risk variants and risk of disease
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7599ed08db3c1e370c210c8789413fdb
https://doi.org/10.1158/1055-9965.22436310
https://doi.org/10.1158/1055-9965.22436310
Autor:
Beatrice Melin, Ulrika Andersson, Tom Børge Johannesen, Preetha Rajaraman, Eivind Hovig, Hilde Langseth, Anna M. Dahlin, Florentin Späth, Carl Wibom
Background: Although glioma etiology is poorly understood in general, growing evidence indicates a genetic component. Four large genome-wide association studies (GWAS) have linked common genetic variants with an increased glioma risk. However, to dat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3b7d0b848dbcb1728b5d74c99fec45d9
https://doi.org/10.1158/1055-9965.c.6515328
https://doi.org/10.1158/1055-9965.c.6515328