Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Carissa Moore"'
Autor:
Anand Pathak, Anand Kolatkar, Amir Ali Talasaz, Julia A. Beaver, Daniel Edelstein, Matthew Ryder, Jerry S.H. Lee, Peter Kuhn, Lea M. Salvatore, Justin Johnson, Seema Singh Bhan, Allison Welsh, David A. Eberhard, James H Godsey, Howard I. Scher, Melanie Hullings, Carolyn Compton, James W. Hicks, Matthew Smalley, Robert L. Grossman, Jonathan W. Simons, Jake Vinson, Gideon M. Blumenthal, David Reese, Sean E. Hanlon, Walt Wells, Rebecca Levine, Phillip G. Febbo, Wendy Royalty, Lauren C. Leiman, Bryan Moy, Hsian-Rong Tseng, Ryan Dittamore, Daniel H. Hovelson, Daniel P. Miller, Anne-Marie Martin, Donald J Johann, Andrew E. Schade, J. Carl Barrett, Hakan Sakul, Carissa Moore, Stella Somiari, Kelli Bramlett, Paul J. Beresford, Andrew Gruen, Reena Philip, John K. Simmons, Angela Silvestro, Jean-Francois Martini, Muneesh Tewari
Publikováno v:
Clinical Pharmacology and Therapeutics
Publikováno v:
The Journal of Immunology. 192:184.23-184.23
Using a mouse model with the tumor suppressor TRAF3 deleted from B cells, we identified Sox5 as a gene strikingly up-regulated in B lymphomas. Sox5 proteins were not detected in normal or premalignant TRAF3-/- B cells even after treatment with B cell
Autor:
Michael Verni, Rodney J. Taylor, Stanislav Zelivianski, Carissa Moore, Dmitriy Kondrikov, Ming Fong Lin
Publikováno v:
Biochimica et biophysica acta. 1539(1-2)
The neuroendocrine (NE) cell is a minor cell population in normal human prostate glands. The number of NE cells is increased in advanced hormone-refractory prostate carcinomas (PCA). The mechanism of increased NE cell population in these advanced tum
Publikováno v:
The Journal of Immunology. 190:188.2-188.2
Myeloid cells, including monocytes, macrophages and granulocytes, are crucial players of innate immunity and inflammation. These cells constitutively or inducibly express a number of receptors of the TNF receptor superfamily and Toll-like receptors (
Publikováno v:
The Journal of Immunology. 188:176.12-176.12
We recently reported that specific deletion of a novel tumor suppressor gene, TRAF3, in B lymphocytes leads to B lymphoma development in mice. Interestingly, B lymphoma development in B-TRAF3-/- mice exhibits a long latency (about 9 months), indicati
Publikováno v:
The Journal of Immunology. 184:138.6-138.6
TRAF3 is a cytoplasmic adaptor protein utilized by the tumor necrosis factor receptor superfamily and Toll-like receptors for signaling. To explore the in vivo functions of TRAF3 in B cells, we recently generated B cell-specific TRAF3-deficient (B-TR