Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Carine Tellier-Lebègue"'
Autor:
Satish K. Tadi, Carine Tellier-Lebègue, Clément Nemoz, Pascal Drevet, Stéphane Audebert, Sunetra Roy, Katheryn Meek, Jean-Baptiste Charbonnier, Mauro Modesti
Publikováno v:
Cell Reports, Vol 17, Iss 2, Pp 541-555 (2016)
In mammalian cells, classical non-homologous end joining (c-NHEJ) is critical for DNA double-strand break repair induced by ionizing radiation and during V(D)J recombination in developing B and T lymphocytes. Recently, PAXX was identified as a c-NHEJ
Externí odkaz:
https://doaj.org/article/c861e7cc7c714c09a07a270f4bfd07e7
Autor:
Pascal Roussel, Stephanie Woerner, Aurélie Gombault, Howard J. Worman, Nathalie Vadrot, Patrick Vicart, Sylvaine Gasparini, Sophie Zinn-Justin, Wikayatou Attanda, Emilie Kondé, Carine Tellier-Lebègue, Cecilia Östlund, Isabelle Duband-Goulet, Constantin T. Craescu, Brigitte Buendia
Publikováno v:
Experimental Cell Research
Experimental Cell Research, Elsevier, 2011, 317 (20), pp.2800-13. ⟨10.1016/j.yexcr.2011.09.012⟩
Experimental Cell Research, 2011, 317 (20), pp.2800-13. ⟨10.1016/j.yexcr.2011.09.012⟩
Experimental Cell Research, Elsevier, 2011, 317 (20), pp.2800-13. ⟨10.1016/j.yexcr.2011.09.012⟩
Experimental Cell Research, 2011, 317 (20), pp.2800-13. ⟨10.1016/j.yexcr.2011.09.012⟩
International audience; Lamins A and C are nuclear intermediate filament proteins expressed in most differentiated somatic cells. Previous data suggested that prelamin A, the lamin A precursor, accumulates in some lipodystrophy syndromes caused by mu
Autor:
Carine Tellier-Lebegue, Eléa Dizet, Emilie Ma, Xavier Veaute, Eric Coïc, Jean-Baptiste Charbonnier, Laurent Maloisel
Publikováno v:
PLoS Genetics, Vol 14, Iss 2, p e1007236 (2018)
[This corrects the article DOI: 10.1371/journal.pgen.1007119.].
Externí odkaz:
https://doaj.org/article/ba4343d950c641c28f528ae2004ba97d
Autor:
Carine Tellier-Lebegue, Eléa Dizet, Emilie Ma, Xavier Veaute, Eric Coïc, Jean-Baptiste Charbonnier, Laurent Maloisel
Publikováno v:
PLoS Genetics, Vol 13, Iss 12, p e1007119 (2017)
Replicative DNA polymerases cannot insert efficiently nucleotides at sites of base lesions. This function is taken over by specialized translesion DNA synthesis (TLS) polymerases to allow DNA replication completion in the presence of DNA damage. In e
Externí odkaz:
https://doaj.org/article/ffa8c3b1930d4be392ff350a6aaa94ce