Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Cardiomegaly/metabolism"'
Autor:
Kyle L. Fulghum, Juliette B. Smith, Julia Chariker, Lauren F. Garrett, Kenneth R. Brittian, Pawel K. Lorkiewicz, Lindsey A. McNally, Shizuka Uchida, Steven P. Jones, Bradford G. Hill, Helen E. Collins
Publikováno v:
Am J Physiol Heart Circ Physiol
Fulghum, K L, Smith, J B, Chariker, J, Garrett, L F, Brittian, K R, Lorkiewicz, P, McNally, L A, Uchida, S, Jones, S P, Hill, B G & Collins, H E 2022, ' Metabolic Signatures of Pregnancy-Induced Cardiac Growth ', American Journal of Physiology: Heart and Circulatory Physiology, vol. 323, no. 1, pp. H146-H164 . https://doi.org/10.1152/ajpheart.00105.2022
Fulghum, K L, Smith, J B, Chariker, J, Garrett, L F, Brittian, K R, Lorkiewicz, P, McNally, L A, Uchida, S, Jones, S P, Hill, B G & Collins, H E 2022, ' Metabolic Signatures of Pregnancy-Induced Cardiac Growth ', American Journal of Physiology: Heart and Circulatory Physiology, vol. 323, no. 1, pp. H146-H164 . https://doi.org/10.1152/ajpheart.00105.2022
The goal of this study was to develop an atlas of the metabolic, transcriptional, and proteomic changes that occur with pregnancy in the maternal heart. Timed pregnancy studies in FVB/NJ mice revealed a significant increase in heart size by day 8 of
Autor:
Richard J. Rodenburg, Ger J.M. Stienen, Heder J de Vries, Hans W.M. Niessen, Liesbeth T. Wintjes, Rob C. I. Wüst
Publikováno v:
Cardiovascular Research, 111(4), 362-372. Oxford University Press
Cardiovascular Research, 111, 4, pp. 362-72
Cardiovascular Research, 111(4), 362-72. Oxford University Press
Wüst, R C I, de Vries, H J, Wintjes, L T, Rodenburg, R J, Niessen, H W M & Stienen, G J M 2016, ' Mitochondrial complex I dysfunction and altered NAD(P)H kinetics in rat myocardium in cardiac right ventricular hypertrophy and failure ', Cardiovascular Research, vol. 111, no. 4, pp. 362-72 . https://doi.org/10.1093/cvr/cvw176
Cardiovascular Research, 111, 362-72
Wust, R C I, de Vries, H J, Wintjes, L T, Rodenburg, R J, Niessen, H W M & Stienen, G J M 2016, ' Mitochondrial complex I dysfunction and altered NAD(P)H kinetics in rat myocardium in cardiac right ventricular hypertrophy and failure ', Cardiovascular Research, vol. 111, no. 4, pp. 362-372 . https://doi.org/10.1093/cvr/cvw176
Cardiovascular Research, 111, 4, pp. 362-72
Cardiovascular Research, 111(4), 362-72. Oxford University Press
Wüst, R C I, de Vries, H J, Wintjes, L T, Rodenburg, R J, Niessen, H W M & Stienen, G J M 2016, ' Mitochondrial complex I dysfunction and altered NAD(P)H kinetics in rat myocardium in cardiac right ventricular hypertrophy and failure ', Cardiovascular Research, vol. 111, no. 4, pp. 362-72 . https://doi.org/10.1093/cvr/cvw176
Cardiovascular Research, 111, 362-72
Wust, R C I, de Vries, H J, Wintjes, L T, Rodenburg, R J, Niessen, H W M & Stienen, G J M 2016, ' Mitochondrial complex I dysfunction and altered NAD(P)H kinetics in rat myocardium in cardiac right ventricular hypertrophy and failure ', Cardiovascular Research, vol. 111, no. 4, pp. 362-372 . https://doi.org/10.1093/cvr/cvw176
Item does not contain fulltext AIMS: In cardiac hypertrophy (CH) and heart failure (HF), alterations occur in mitochondrial enzyme content and activities but the origin and implications of these changes for mitochondrial function need to be resolved.
Autor:
Tatiana Krebs, Allison Felley, Christophe Albert Montessuit, Jean-Claude Perriard, Ellen Aasum, Anna Bogdanova, Renata Windak, Wilhelm Krek, Thierry Pedrazzini, Jaya Krishnan, Malgorzata Tokarska-Schlattner, Marianne Suter, Terje S. Larsen, Evelyne Perriard
Publikováno v:
Cell metabolism
Cell Metabolism, Vol. 9, No 6 (2009) pp. 512-524
Cell Metabolism
Cell Metabolism, Elsevier, 2009, 9 (6), pp.512-524. ⟨10.1016/j.cmet.2009.05.005⟩
Cell Metabolism, Vol. 9, No 6 (2009) pp. 512-524
Cell Metabolism
Cell Metabolism, Elsevier, 2009, 9 (6), pp.512-524. ⟨10.1016/j.cmet.2009.05.005⟩
Development of cardiac hypertrophy and progression to heart failure entails profound changes in myocardial metabolism, characterized by a switch from fatty acid utilization to glycolysis and lipid accumulation. We report that hypoxia-inducible factor
Autor:
Fang Zhang, F. Donelson Smith, Alexandra C. Newton, Dario Diviani, Lorene K. Langeberg, John D. Scott, Joseph Soughayer, Michael R. Bristow, Maya T. Kunkel, Graeme K. Carnegie, Benjamin S. Pedroja
Publikováno v:
Molecular Cell, vol. 32, no. 2, pp. 169-179
Elevated catecholamines in the heart evoke transcriptional activation of the Myocyte Enhancer Factor (MEF) pathway to induce a cellular response known as pathological myocardial hypertrophy. We have discovered that the A-Kinase Anchoring Protein (AKA
Autor:
Mireia Jové, Ricardo Rodríguez-Calvo, Anna Planavila, Manuel Vázquez-Carrera, Walter Wahli, Liliane Michalik, Juan C. Laguna
Publikováno v:
Cardiovascular Research, vol. 65, no. 4, pp. 832-841
CARDIOVASC RES
r-FSJD. Repositorio Institucional de Producción Científica de la Fundació Sant Joan de Déu
instname
r-FSJD: Repositorio Institucional de Producción Científica de la Fundació Sant Joan de Déu
Fundació Sant Joan de Déu
CARDIOVASC RES
r-FSJD. Repositorio Institucional de Producción Científica de la Fundació Sant Joan de Déu
instname
r-FSJD: Repositorio Institucional de Producción Científica de la Fundació Sant Joan de Déu
Fundació Sant Joan de Déu
OBJECTIVE: Peroxisome proliferator-activated receptor beta/delta (PPARbeta/delta) is the predominant PPAR subtype in cardiac cells and plays a prominent role in the regulation of cardiac lipid metabolism. However, the role of PPARbeta/delta activator
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e33838777d087243b40959f69d7e6a1f
https://serval.unil.ch/resource/serval:BIB_EBA9B8610022.P001/REF.pdf
https://serval.unil.ch/resource/serval:BIB_EBA9B8610022.P001/REF.pdf
Autor:
Robberecht, Patrick, Waelbroeck, Magali, Claeys, Magda, Nguyen Huu, Anh, Chatelain, Pierre, Christophe, Jean
Publikováno v:
Molecular pharmacology, 21 (3
The effects of the thyroid state and of aortic stenosis on muscarinic cholinergic binding sites in heart membranes were compared (with proper controls) by simultaneously determining total and high-affinity binding sites and estimating low-affinity bi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______2101::d7b4255c44ba2ee16f8af685d3e93978
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/148249
http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/148249
Autor:
Patrick Robberecht, Magali Waelbroeck, Jean Christophe, Anh Nguyen Huu, Magda Claeys, Pierre Chatelain
Publikováno v:
European journal of pharmacology, 91 (2-3
The effect of phenylhydrazine-induced heart hypertrophy on muscarinic cholinergic binding sites was studied in heart membranes by determining total (with [3H]NMS) and high-affinity (with [3H]Oxo-M) binding sites and estimating low-affinity binding si