Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Candace M. Adamo"'
Publikováno v:
The Journal of Applied Laboratory Medicine. 2:822-824
Several sensitive low-abundance urine protein biomarkers have emerged for the diagnosis of acute kidney injury (AKI)2, but preanalytical factors can negatively impact the accuracy of biomarker testing. In this study, we observed the effect of a hydro
Autor:
Justin M. Percival, Candace M. Adamo, Joseph A. Beavo, Stanley C. Froehner, Nicholas P. Whitehead, Marvin E. Adams
Publikováno v:
The Journal of Pathology. 228:77-87
Duchenne muscular dystrophy (DMD) is the most common form of muscular dystrophy caused by mutations in the dystrophin gene. Loss of dystrophin initiates a progressive decline in skeletal muscle integrity and contractile capacity which weakens respira
Autor:
Bernard Lassègue, Bonnie Seidel-Rogol, Srinivasan Raju Datla, Candace M. Adamo, Lily Pounkova, Holly C. Williams, Kathy K. Griendling, Alejandra San Martin, James E. Bear
Rationale: The type I subclass of coronins, a family of actin-binding proteins, regulates various actin-dependent cellular processes, including migration. However, the existence and role of coronins in vascular smooth muscle cell (VSMC) migration has
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d79e9602061045a1745c297368dc6c61
Autor:
Monte S Willis, Dao-Fu Dai, Sergei D. Rybalkin, Stanley C. Froehner, Joseph A. Beavo, Justin M. Percival, Elina Minami, Candace M. Adamo, Enrico Patrucco
Publikováno v:
BMC Pharmacology
Recent results show that the classic PDE5 inhibitor, Viagra® (sildenafil), can ameliorate much of the cardiac pathology resulting from several different forms of cardiac damage. These results have elicited great interest in the use of this drug as a
Publikováno v:
Phosphodiesterases as Drug Targets ISBN: 9783642179686
Duchenne muscular dystrophy (DMD) is a devastating and ultimately fatal disease characterized by progressive muscle wasting and weakness. DMD is caused by the absence of a functional dystrophin protein, which in turn leads to reduced expression and m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ced29346c1f298618a9dd8a3cb5cad5d
https://doi.org/10.1007/978-3-642-17969-3_14
https://doi.org/10.1007/978-3-642-17969-3_14
Autor:
Justin M. Percival, Dao-Fu Dai, Elina Minami, Stanley C. Froehner, Joseph A. Beavo, Candace M. Adamo, Monte S. Willis, Enrico Patrucco
Duchenne muscular dystrophy (DMD) is a progressive and fatal genetic disorder of muscle degeneration. Patients with DMD lack expression of the protein dystrophin as a result of mutations in the X-linked dystrophin gene. The loss of dystrophin leads t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::acdf4a3d8ec172bf24543f047da58b99
https://europepmc.org/articles/PMC2973894/
https://europepmc.org/articles/PMC2973894/
Autor:
Joseph A. Beavo, Nicholas P. Whitehead, Candace M. Adamo, Justin M. Percival, Marvin E. Adams, Stanley C. Froehner
Publikováno v:
Neuromuscular Disorders. 22:848
Until genetic correction approaches are routinely available for treatment of muscular dystrophies, treatments that extend longevity and improve quality of life will be important alternatives. Based on our studies of one isoform of nitric oxide syntha