Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Camilla Udjus"'
Autor:
Camilla Udjus, Bente Halvorsen, Xiang Yi Kong, Ellen Lund Sagen, Marita Martinsen, Kuan Yang, Else Marit Løberg, Geir Christensen, Ole Henning Skjønsberg, Karl‐Otto Larsen
Publikováno v:
Physiological Reports, Vol 12, Iss 14, Pp n/a-n/a (2024)
Abstract Inflammation through activation of caspase‐1, seems to play a role in pulmonary hypertension induced by alveolar hypoxia. Whether alveolar hypoxia induces caspase‐1‐mediated inflammation and influx of leukocytes in other organs than th
Externí odkaz:
https://doaj.org/article/f25c7b58ac1744ec9d818268d473829d
Autor:
Camilla Udjus, Ivar Sjaastad, Ulla Hjørnholm, Torbjørn K. Tunestveit, Pavel Hoffmann, Alexis Hinojosa, Emil K. S. Espe, Geir Christensen, Ole H. Skjønsberg, Karl‐Otto Larsen, Morten Rostrup
Publikováno v:
Physiological Reports, Vol 10, Iss 3, Pp n/a-n/a (2022)
Abstract Mountain climbing at high altitude implies exposure to low levels of oxygen, low temperature, wind, physical and psychological stress, and nutritional insufficiencies. We examined whether right ventricular (RV) and left ventricular (LV) myoc
Externí odkaz:
https://doaj.org/article/20eaa3225b14408db9e1771233728200
Autor:
Ivar Sjaastad, Ole Henning Skjønsberg, Pavel Hoffmann, Torbjørn Tunestveit, Ulla Hjørnholm, Morten Rostrup, Karl-Otto Larsen, Camilla Udjus
Publikováno v:
Clinical respiratory physiology, exercise and functional imaging.
Background: A zest for mountain climbing has developed, and the number of deaths related to this activity has risen. Hypobaric hypoxia, causing hypoxemia and tissue hypoxia, triggers numerous responses in the body such as acute mountain sickness, pul
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0047eca01bc20d94a84893439aed4f35
https://doi.org/10.1183/13993003.congress-2020.2561
https://doi.org/10.1183/13993003.congress-2020.2561
Autor:
Geir Christensen, Bente Halvorsen, Karl-Otto Larsen, Camilla Udjus, Pål Aukrust, Ole Henning Skjønsberg
Publikováno v:
Pulmonary hypertension.
Background: Pulmonary hypertension is a serious complication to chronic lung diseases, often with alveolar hypoxia. Mechanisms for hypoxia-induced pulmonary hypertension are suggested to be initial vasoconstriction, followed by inflammation, prolifer
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7ce069b549331f31c163bb0437a16997
https://doi.org/10.1183/13993003.congress-2019.pa5049
https://doi.org/10.1183/13993003.congress-2019.pa5049
Autor:
Ivar Sjaastad, Dina Behmen, Ole Henning Skjønsberg, Karl-Otto Larsen, Geir Christensen, Fadila Telarevic Cero, Bård Andre Bendiksen, Else M. Løberg, Pål Aukrust, Emil K. S. Espe, Cathrine R. Carlson, Arne Yndestad, Camilla Udjus, Bente Halvorsen
Lung diseases with hypoxia are complicated by pulmonary hypertension, leading to heart failure and death. No pharmacological treatment exists. Increased proinflammatory cytokines are found in hypoxic patients, suggesting an inflammatory pathogenesis.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b944692bbc5c56af81ce1a19bddf5f07
http://hdl.handle.net/10852/75429
http://hdl.handle.net/10852/75429
Autor:
Pål Aukrust, Ole Henning Skjønsberg, Emil K. S. Espe, Fadila Telarevic Cero, Cathrine R. Carlson, Arne Yndestad, Karl-Otto Larsen, Bård Andre Bendiksen, Ivar Sjåstad, Else Marit Løberg, Camilla Udjus, Geir Christensen, Bente Halvorsen
Publikováno v:
Pulmonary Circulation and Pulmonary Vascular Disease.
Background: Pulmonary hypertension is a severe complication to chronic lung diseases, often with alveolar hypoxia. Inflammasomes are large multiprotein oligomers consisting of receptor molecules (e.g. NLR), an adaptor protein (ASC) and the effector e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a89e3baef47e603c060d8c5a3f2a26ac
https://doi.org/10.1183/1393003.congress-2017.pa2373
https://doi.org/10.1183/1393003.congress-2017.pa2373
Autor:
Ivar Sjaastad, Maria Belland Olsen, Pål Aukrust, Karl Otto Larsen, Vigdis Hillestad, Ole Henning Skjønsberg, Fadila Telarevic Cero, Camilla Udjus, Arne Yndestad, Else Marit Løberg, Geir Christensen
Publikováno v:
4.3 Pulmonary Circulation and Pulmonary Vascular Disease.
Background: Inflammasomes are large multiprotein oligomers consisting of a receptor (such as NLRP3), the adaptor protein (ASC) and caspase-1. Assembly of the inflammasome activates caspase-1 which cleaves interleukin (IL)-18 and IL-1β to their bioac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::367201a6d12a87a388f4be03c3ab0c27
https://doi.org/10.1183/13993003.congress-2015.pa4908
https://doi.org/10.1183/13993003.congress-2015.pa4908