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The cardiomyocyte phenotypic switch from a proliferative to terminally differentiated state results in the loss of regenerative potential of the mammalian heart shortly after birth. Yet, the molecular mechanisms that regulate this critical developmen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1704ada5cd69e3f143f67d0df5c4dc5e
https://hdl.handle.net/11541.2/32645
https://hdl.handle.net/11541.2/32645
