Zobrazeno 1 - 10
of 11
pro vyhledávání: '"C. J. Shorrock"'
Autor:
W. D. W. Rees, C. J. Shorrock
Publikováno v:
Gut. 33:164-169
The effect of 28 days' continuous administration of oral indomethacin on gastroduodenal morphology, gastric mucosal blood flow, and gastric mucosal prostaglandin E2 (PGE2) metabolism in man was studied to define further the mechanisms of mucosal inju
Publikováno v:
Gut. 37:509-511
From 1979 to 1985 2435 patients having had transient ischaemic attacks (TIAs) or minor ischaemic strokes, were enrolled in the UK TIA trial and were randomised to receive either aspirin 300 mg, daily or aspirin 1200 mg or placebo. Analysis of reporte
Autor:
C. J. Shorrock
Publikováno v:
Current Opinion in Gastroenterology. 6:888-890
Autor:
C J, Shorrock, W D, Rees
Publikováno v:
The Journal of rheumatology. Supplement. 36
Autor:
C J, Shorrock, W D, Rees
Publikováno v:
The Journal of rheumatology. Supplement. 36
Publikováno v:
Gut. 31(1)
Acid and alkali secretion have been examined together with prostaglandin E2 production in response to two mucosal protective drugs, colloidal bismuth subcitrate and sucralfate. Doses of colloidal bismuth subcitrate in the therapeutic range (120 and 1
Publikováno v:
Gut. 31:958-959
Publikováno v:
Gut. 30:917-921
The ulcer healing and cytoprotective properties of colloidal bismuth (De-Nol) are well established although its mode of action is unclear. We have examined the action of bismuth subcitrate, the active ingredient of De-Nol, on gastroduodenal bicarbona
Publikováno v:
Digestive Diseases and Sciences. 34:1016-1020
The protective and ulcer healing properties of prostaglandins are well established. We have explored the possible mode of action of enprostil, a synthetic dehydroprostaglandin E2, on amphibian gastroduodenal mucosal bicarbonate secretion in vitro and
Autor:
C. J. Shorrock, W. D. W. Rees
Publikováno v:
Scandinavian journal of rheumatology. Supplement. 78
All non-steroidal anti-inflammatory drugs (NSAIDs) used in the treatment of rheumatic diseases may cause gastrointestinal mucosal injury. The mechanisms by which these agents damage mucosa are not fully understood, although, reduction of mucosal defe