Zobrazeno 1 - 10
of 30
pro vyhledávání: '"C. F. Rothe"'
Publikováno v:
American Journal of Physiology-Gastrointestinal and Liver Physiology. 261:G539-G547
We tested the hypotheses that the hepatic venule pressures (Phv), just downstream from the hepatic sinusoids, are closely similar (less than 2 mmHg) either to the portal venous pressure (Ppv), indicating a high hepatic venous resistance, or to the in
Autor:
Monica Gaddis, C. F. Rothe
Publikováno v:
Circulation. 81:360-368
After a change in cardiac output, the magnitude of potential blood volume redistribution was investigated in 10 dogs anesthetized with chloralose. All of the venous return was pumped into a reservoir, using servocontrolled pumps to maintain fixed sup
Autor:
C. F. Rothe, R. Maass-Moreno
Publikováno v:
The American journal of physiology. 262(1 Pt 1)
We tested the hypothesis that the larger (greater than 2 mm ID) hepatic veins are the primary site of the portal-to-caval venous pressure gradient in the dog. Double-lumen catheters were inserted through the caval wall into hepatic veins of pentobarb
Autor:
R. Maass-Moreno, C. F. Rothe
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 260:1-1
Pages H254–H259: R. Maass-Moreno and C. F. Rothe. “Carotid baroreceptor control of liver and spleen volume in cats.” Page H254: The sentence beginning on line 11 of the abstract should read: In vagotomized cats, increasing Pcs by 100 mmHg cause
Autor:
C. F. Rothe
Publikováno v:
Archives of Internal Medicine. 146:977-982
Publikováno v:
Circulation Research. 47:551-558
To test whether the pressure-volume relationship of the canine small intestinal vasculature is linear over the normal range of portal venous pressures (5 to 35 mm Hg), we used two methods to measure volume: (1) the integral of inflow minus outflow (I
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 251:H164-H170
Hypercapnic stimulation of the brain may account for some of the decrease in vascular capacitance (venoconstriction) seen with whole-body hypercapnia. Six mongrel dogs were anesthetized with alpha-chloralose and paralyzed with pancuronium bromide. Th
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 242:H1000-H1009
Active changes in hepatic capacitance were studied in pump-perfused dog livers during hepatic nerve stimulation or during intrahepatic arterial infusion of histamine (0.01-1 mg/l) or epinephrine (0.05 mg/l). Hepatic nerve stimulation at 5 pulses/s (p
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 251:H857-H862
Three experimental series using 22 acutely splenectomized mongrel dogs were completed to 1) compare fibrillation (Fib) and acetylcholine (ACh) injection as methods to stop the heart for the mean circulatory filling pressure (Pmcf) maneuver, and 2) te
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 249:H1061-H1069
The magnitude of vascular capacitance change induced by hypercapnia, hypoxia, or hypoxic hypercapnia was estimated during the administration of experimental gas mixtures to anesthetized dogs for 25 min. Mean circulatory filling pressure (Pcf) was det