Zobrazeno 1 - 10
of 11
pro vyhledávání: '"C. Brad Wilson"'
Publikováno v:
PLoS ONE, Vol 11, Iss 9, p e0160923 (2016)
Post-traumatic stress disorder (PTSD) is a trauma and stressor-related disorder that results in a prolonged stress response. It is associated with increased oxidative stress and inflammation in the prefrontal cortex (PFC) and hippocampus (HC). The on
Externí odkaz:
https://doaj.org/article/98ecd31867804db39bf7fe5cc4f04a52
Publikováno v:
PLoS ONE, Vol 9, Iss 2, p e89104 (2014)
Post-Traumatic Stress Disorder (PTSD) can develop in response to a traumatic event involving a threat to life. To date, no diagnostic biomarkers have been identified for PTSD. Recent research points toward physiological abnormalities in the hypothala
Externí odkaz:
https://doaj.org/article/1547552db1d44f37a59a05a8c62e2c24
Autor:
C Brad Wilson, Leslie D McLaughlin, Anand Nair, Philip J Ebenezer, Rahul Dange, Joseph Francis
Publikováno v:
PLoS ONE, Vol 8, Iss 10, p e76146 (2013)
This study sought to analyze specific pathophysiological mechanisms involved in the progression of post-traumatic stress disorder (PTSD) by utilizing an animal model. To examine PTSD pathophysiology, we measured damaging reactive oxygen species and i
Externí odkaz:
https://doaj.org/article/69214167c2f541769a12abc6a0750cbf
Publikováno v:
Behavioural Brain Research. 268:72-80
Reactive oxygen species (ROS) and pro-inflammatory cytokines (PIC) are upregulated in post-traumatic stress disorder (PTSD). Histone deacetylase inhibitors (HDACi) modify genetic transcription and can diminish ROS and PIC escalation. They can also mo
Publikováno v:
Military medicine. 181(11)
Recent conflicts have contributed to an escalation in combat-related psychiatric disorders, including post-traumatic stress disorder (PTSD). Although technological advances have increased the speed from which battlefield injuries reach definitive car
Autor:
Philip J. Ebenezer, C. Brad Wilson, Rahul B. Dange, Joseph Francis, Leslie D. McLaughlin, Anand R. Nair
Publikováno v:
Comprehensive Guide to Post-Traumatic Stress Disorder ISBN: 9783319086132
We sought to analyze specific pathophysiological mechanisms involved in the progression of posttraumatic stress disorder (PTSD) by utilizing an animal model. To examine PTSD pathophysiology, we measured reactive oxygen species and inflammatory cytoki
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0742468f28494b29a6b6504ab3bcc82b
https://doi.org/10.1007/978-3-319-08613-2_87-1
https://doi.org/10.1007/978-3-319-08613-2_87-1
Autor:
Rahul B. Dange, Thomas Shaak, Leslie D. McLaughlin, Joseph Francis, Joseph Harre, Philip J. Ebenezer, Anand R. Nair, C. Brad Wilson, David M. Diamond
Publikováno v:
Frontiers in Behavioral Neuroscience, Vol 8 (2014)
Frontiers in Behavioral Neuroscience
Frontiers in Behavioral Neuroscience
Serotonin (5-HT), norepinephrine (NE), and other neurotransmitters are modulated in post-traumatic stress disorder (PTSD). In addition, pro-inflammatory cytokines (PIC) are elevated during the progression of the disorder. Currently, the only approved
Publikováno v:
PLoS ONE
PLoS ONE, Vol 9, Iss 2, p e89104 (2014)
PLoS ONE, Vol 9, Iss 2, p e89104 (2014)
Post-Traumatic Stress Disorder (PTSD) can develop in response to a traumatic event involving a threat to life. To date, no diagnostic biomarkers have been identified for PTSD. Recent research points toward physiological abnormalities in the hypothala
Autor:
Joseph Francis, Leslie D. McLaughlin, Rahul B. Dange, Anand R. Nair, Philip J. Ebenezer, C. Brad Wilson
Publikováno v:
PLoS ONE
PLoS ONE, Vol 8, Iss 10, p e76146 (2013)
PLoS ONE, Vol 8, Iss 10, p e76146 (2013)
This study sought to analyze specific pathophysiological mechanisms involved in the progression of post-traumatic stress disorder (PTSD) by utilizing an animal model. To examine PTSD pathophysiology, we measured damaging reactive oxygen species and i
Publikováno v:
PLoS ONE
PLoS ONE, Vol 11, Iss 9, p e0160923 (2016)
PLoS ONE, Vol 11, Iss 9, p e0160923 (2016)
Post-traumatic stress disorder (PTSD) is a trauma and stressor-related disorder that results in a prolonged stress response. It is associated with increased oxidative stress and inflammation in the prefrontal cortex (PFC) and hippocampus (HC). The on