Zobrazeno 1 - 10
of 260
pro vyhledávání: '"C S Lieber"'
Publikováno v:
Journal of Lipid Research, Vol 27, Iss 10, Pp 1073-1083 (1990)
To study the mechanism of alcoholic hypertriglyceridemia, baboons were pair-fed liquid diets containing 50% of energy as ethanol or as additional carbohydrate for 5-16 months. Alcohol-fed animals developed hypertriglyceridemia and early stages of alc
Externí odkaz:
https://doaj.org/article/d38c588e8550482da931afe5a0d51864
Autor:
E Baraona, C S Lieber
Publikováno v:
Journal of Lipid Research, Vol 20, Iss 3, Pp 289-315 (1979)
Alcohol promotes accumulation of fat in the liver mainly by substitution of ethanol for fatty acids as the major hepatic fuel. The degree of lipid accumulation depends on the supply of dietary fat. Progressive alteration of the mitochondria, which oc
Externí odkaz:
https://doaj.org/article/4a314abde2b14a0eb1e99cac882dab24
Publikováno v:
Journal of Lipid Research, Vol 25, Iss 8, Pp 813-820 (1984)
To study the effects of alcoholic liver injury on the ability of ethanol to promote hepatic fat accumulation and hyperlipemia, baboons were pair-fed liquid diets containing 50% of energy either as ethanol or as additional carbohydrate (controls) for
Externí odkaz:
https://doaj.org/article/d23552b74a4c42f2a474f76f5373106d
Autor:
C. S. Lieber
Publikováno v:
Chemical and Biological Aspects of Drug Dependence ISBN: 9780429260629
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::cc43fd6198148692ac971ad6695739f2
https://doi.org/10.1201/9780429260629-15
https://doi.org/10.1201/9780429260629-15
Autor:
A J, Garro, C S, Lieber
Publikováno v:
Annual Review of Pharmacology and Toxicology. 30:219-249
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::257567758a3fe215361b4366fdeefa5f
https://doi.org/10.1146/annurev.pa.30.040190.001251
https://doi.org/10.1146/annurev.pa.30.040190.001251
Autor:
C S, Lieber
Publikováno v:
Roczniki Akademii Medycznej w Bialymstoku (1995). 50
Fifty years ago the dogma prevailed that alcohol was not toxic to the liver and that alcoholic liver disease was exclusively a consequence of nutritional deficiencies. We showed, however, that liver pathology developed even in the absence of malnutri
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::493e3736487c84cb085cea6a2cfbac8c
https://pubmed.ncbi.nlm.nih.gov/16363067
https://pubmed.ncbi.nlm.nih.gov/16363067
Publikováno v:
Alcoholism, clinical and experimental research. 25(9)
Hepatotoxicity of ethanol is increased by beta-carotene in both rodents and nonhuman primates. Furthermore, in smokers who are also drinkers, beta-carotene increases the incidence of pulmonary cancer. The hepatotoxicity was associated with proliferat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::ed457651af5b1eaf09a3ea43d7ea6aaf
https://pubmed.ncbi.nlm.nih.gov/11584158
https://pubmed.ncbi.nlm.nih.gov/11584158