Zobrazeno 1 - 10
of 29
pro vyhledávání: '"C R Pullinger"'
Autor:
C R Pullinger, M J Malloy, A K Shahidi, M Ghassemzadeh, P Duchateau, J Villagomez, J Allaart, J P Kane
Publikováno v:
Journal of Lipid Research, Vol 38, Iss 9, Pp 1833-1840 (1997)
Apolipoprotein C-III (apoC-III) is a major protein component of very low density lipoproteins (VLDL), chylomicrons, and a minor component of high density lipoproteins (HDL). Studies of naturally occurring human variants of apoC-III will help in addin
Externí odkaz:
https://doaj.org/article/cac5d33133374fccab85276edb908faf
Autor:
R van Antwerpen, G C Chen, C R Pullinger, J P Kane, M LaBelle, R M Krauss, C Luna-Chavez, T M Forte, J C Gilkey
Publikováno v:
Journal of Lipid Research, Vol 38, Iss 4, Pp 659-669 (1997)
Cryo-electron microscopy was used to analyze the structure of low density lipoprotein from normolipidemic subjects (N-LDL), phospholipid-depleted N-LDL (PD-LDL), small dense LDL from hypertriglyceridemic subjects (SD-LDL), and reconstituted discoidal
Externí odkaz:
https://doaj.org/article/d9c911da315a4b7c85d4e4604024e50c
Publikováno v:
Journal of Lipid Research, Vol 25, Iss 12, Pp 1358-1367 (1984)
In rat hepatocytes freshly isolated from donor rats at different times of the day, the rates of lipogenesis (de novo fatty acid synthesis) varied with a diurnal periodicity. The maximal rate occurred approximately 5 hr after the end of the normal 8-h
Externí odkaz:
https://doaj.org/article/503f5be511f441fbbcb78f220f232293
Autor:
C R Pullinger, G F Gibbons
Publikováno v:
Journal of Lipid Research, Vol 24, Iss 10, Pp 1321-1328 (1983)
The true rate of sterol synthesis in liver cells was determined by measurement of the weight of desmosterol produced over a given time period during incubations in the presence of triparanol. The simultaneous presence of tritiated water (3H2O) during
Externí odkaz:
https://doaj.org/article/e2b65c104a96480eb29932316201f18c
Publikováno v:
Journal of Lipid Research, Vol 30, Iss 7, Pp 1065-1077 (1989)
The objective of this study was to establish conditions whereby apoB secreted from HepG2 cells could be regulated over a wide range, and to determine whether changes of output were correlated with the level of apoB mRNA. The presence of oleate (compl
Externí odkaz:
https://doaj.org/article/33b13bd9911a4468bd08ec39eb911077
Autor:
John C. Gilkey, M LaBelle, R M Krauss, John P. Kane, G C Chen, C R Pullinger, C Luna-Chavez, T M Forte, R. Van Antwerpen
Publikováno v:
Journal of Lipid Research, Vol 38, Iss 4, Pp 659-669 (1997)
Cryo-electron microscopy was used to analyze the structure of low density lipoprotein from normolipidemic subjects (N-LDL), phospholipid-depleted N-LDL (PD-LDL), small dense LDL from hypertriglyceridemic subjects (SD-LDL), and reconstituted discoidal
Autor:
C R, Pullinger, D, Gaffney, M M, Gutierrez, M J, Malloy, V N, Schumaker, C J, Packard, J P, Kane
Publikováno v:
Journal of lipid research. 40(2)
Familial ligand-defective apolipoprotein B (apoB) is a group of disorders caused by mutations in the apoB gene. In this report the R3531C mutation is characterized further using a monoclonal antibody MB19/dynamic laser light scattering technique to m
Autor:
M S, Nauck, H, Nissen, M M, Hoffmann, J, Herwig, C R, Pullinger, M, Averna, J, Geisel, H, Wieland, W, März
Publikováno v:
Clinical chemistry. 44(7)
Familial apolipoprotein (apo) CII deficiency is a rare autosomal recessive inborn error of metabolism clinically resembling lipoprotein lipase deficiency. A number of mutations of the apo CII gene are known to date; they are located in the promoter r
Autor:
P N, Duchateau, C R, Pullinger, R E, Orellana, S T, Kunitake, J, Naya-Vigne, P M, O'Connor, M J, Malloy, J P, Kane
Publikováno v:
The Journal of biological chemistry. 272(41)
In this study, we have identified and characterized a new protein present in human high density lipoprotein that we have designated apolipoprotein L. Using a combination of liquid-phase isoelectrophoresis and high resolution two-dimensional gel elect
Publikováno v:
Biochemical Journal. 183:309-315
Carbon monoxide inhibited the removal of C-32 of dihydrolanosterol (I), but not of its metabolites 5 alpha-lanost-8-ene-3 beta,32-diol (II) and 3 beta-hydroxy-5 alpha-lanost-8-en-32-al (III). It appears therefore that cytochrome P-450 is a component