Zobrazeno 1 - 10
of 86
pro vyhledávání: '"C L, CONLEY"'
Autor:
N. U. Bang, S. J. Shattil, T. G. Evans, C. R. Valeri, F. Pompei, S. C. Cramer, S. S. Buys, Shukri F. Khuri, J. N. George, G. J. Schiller, C. L. Conley, H. MacGregor, G. M. Rodgers
Publikováno v:
New England Journal of Medicine. 324:1670-1672
Autor:
C. L. Conley
Publikováno v:
JAMA: The Journal of the American Medical Association. 263:2481-2483
Autor:
C L, Conley
Publikováno v:
JAMA. 263(18)
Publikováno v:
Blood. 49:563-571
Systemic mastocytosis occurred as a fatal event in a patient with long- standing polycythemia vera. The patient had been treated over the course of 21 yr with radioactive phosphorus. Possible relationships between mastocytosis and polycythemia vera,
Publikováno v:
Annals of the New York Academy of Sciences. 232:125-134
Publikováno v:
Medicine. 59:323-334
Among 320 relatives of 28 patients with chronic lymphocytic leukemia (CLL), 4 had CLL, 1 had lymphosarcoma, and 2 other adults had leukemia of unknown type. Autoimmune disease including hyperthyroidism, pernicious anemia, rheumatoid arthritis and sys
Publikováno v:
Blood. 49:563-571
Systemic mastocytosis occurred as a fatal event in a patient with long- standing polycythemia vera. The patient had been treated over the course of 21 yr with radioactive phosphorus. Possible relationships between mastocytosis and polycythemia vera,
Publikováno v:
Journal of Clinical Investigation. 46:1795-1811
Hemoglobin C is less soluble than hemoglobin A in red cells, in hemolysates, and in dilute phosphate buffer. Its relative insolubility may be explained by electrostatic interactions between positively charged beta6-lysyl groups and negatively charged
Publikováno v:
Journal of Experimental Medicine. 91:123-133
A fraction of globulin was prepared from human plasma which was deficient in prothrombin, thrombin, fibrinogen, plasma thromboplastin, and accelerator globulin. The preparation of globulin contained considerable potential proteolytic activity which c
Publikováno v:
Blood. 21:261-281
Hereditary persistence of fetal hemoglobin is an anomaly of hemoglobin production apparently caused by a mutant gene that inhibits synthesis of hemoglobins A and A2. Alkali-resistant hemoglobin indistinguishable from hemoglobin F of umbilical cord bl