Zobrazeno 1 - 10
of 14
pro vyhledávání: '"C G, Yeh"'
Autor:
Yee Chao, C. G. Yeh, M. B. Cardic, Amitesh Roy, S. R. Park, Y-K. Kang, M. Kujundzic, Josep Tabernero, Francisco Javier Ramos, A. de Gramont, Charlotte Rees, David Cunningham, Ho-Yeong Lim, Li-Tzong Chen
Publikováno v:
Annals of Oncology. 24:1567-1573
Background PEP02 is a novel highly stable liposomal nanocarrier formulation of irinotecan. This randomized phase II study evaluated the efficacy and safety of single agent PEP02 compared with irinotecan or docetaxel in the second-line treatment of ad
Publikováno v:
The Journal of Immunology. 158:2872-2881
A chimeric gene was constructed from the genes coding for the human complement regulatory proteins, membrane cofactor protein (CD46) and decay-accelerating factor (CD55). The recombinant chimeric gene was transfected into Chinese hamster ovary cells.
Publikováno v:
Journal of the American Society of Nephrology. 5:1888-1894
Complement is a major mediator of tissue injury in several types of glomerulonephritis. However, no therapeutic agents that inhibit complement activation are available for human use. sCR1 (TP10, BRL 55736) is a recombinant, soluble human complement r
Publikováno v:
The Journal of Immunology. 149:1723-1728
We examined the role of C activation in ischemia reperfusion injury by inhibiting C activation in a rat model of mesenteric arterial occlusion. In anesthetized rats, 60 min of mesenteric arterial occlusion was followed by 3 h of reperfusion. PBS alon
Autor:
R Rabinovici, C G Yeh, L M Hillegass, D E Griswold, M J DiMartino, J Vernick, K L Fong, G Feuerstein
Publikováno v:
The Journal of Immunology. 149:1744-1750
C receptor-1 is a protein involved in the regulation of C3 and C5-convertases. Recombinant human soluble C receptor-1 has recently been produced and shown to reduce infarct size in a rat model of myocardial ischemia/reperfusion injury. The present st
Publikováno v:
The Journal of Immunology. 148:3086-3092
Acute lung injury produced by deposition of IgA immune complexes is complement-dependent, neutrophil-independent, oxygen radical-mediated, and may be a result of the formation of the hydroxyl radical (HO) generated directly or indirectly from activat
Publikováno v:
The Journal of Immunology. 148:1479-1485
Complement activation is an important step for triggering of acute inflammatory reactions. Soluble human recombinant complement receptor type 1 (sCR1) blocks complement activation by both classical and alternative pathways. In addition to glycogen-in
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 158(6)
A chimeric gene was constructed from the genes coding for the human complement regulatory proteins, membrane cofactor protein (CD46) and decay-accelerating factor (CD55). The recombinant chimeric gene was transfected into Chinese hamster ovary cells.
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 149(5)
We examined the role of C activation in ischemia reperfusion injury by inhibiting C activation in a rat model of mesenteric arterial occlusion. In anesthetized rats, 60 min of mesenteric arterial occlusion was followed by 3 h of reperfusion. PBS alon
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 148(10)
Acute lung injury produced by deposition of IgA immune complexes is complement-dependent, neutrophil-independent, oxygen radical-mediated, and may be a result of the formation of the hydroxyl radical (HO) generated directly or indirectly from activat