Abstract 523: Sodium Nitrite Therapy Augments Ischemia-Induced Neovascularization and Blood Flow Recovery in Different Models of Hypertension

Autor: Ali Amin, Sookyoung Choi, Y Osman-Elazeik, N Badr El-Din, C G Kevil, G L Navar, P Kadowitz, M Trebak, Khalid Matrougui

Publikováno v: Hypertension. 60

Background: Arterial hypertension is a major risk factor that can lead to exacerbation of peripheral vascular disease due, in part, to endothelial dysfunction. Because sodium nitrite (SN) can be converted to nitric oxide (NO), which counteracts endot

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::8b890d20be0aa6700061987ad15c6752
https://doi.org/10.1161/hyp.60.suppl_1.a523

Self‐Assembled Aza‐Boron‐Dipyrromethene‐Based H2S Prodrug for Synergistic Ferroptosis‐Enabled Gas and Sonodynamic Tumor Therapies.

Autor: Zhao, Jiajia¹ (AUTHOR), Bian, Erbao¹ (AUTHOR), Zhang, Renwu¹ (AUTHOR), Xu, Tao² (AUTHOR), Nie, Yang¹ (AUTHOR), Wang, Linqi¹ (AUTHOR), Jin, Gui¹ (AUTHOR), Xie, Han¹ (AUTHOR), Xiang, Huijing³ (AUTHOR) xianghuijing@shu.edu.cn, Chen, Yu^3,4,5 (AUTHOR) chenyuedu@shu.edu.cn, Wu, Dejun¹ (AUTHOR) wudj2023@163.com

Publikováno v: Advanced Science. 8/14/2024, Vol. 11 Issue 30, p1-14. 14p.

H2S Increases Blood Pressure via Activation of L‐Type Calcium Channels with Mediation by HS^• Generated from Reactions with Oxyhemoglobin.

Autor: Liu, Taiming¹ (AUTHOR), Zhang, Meijuan¹ (AUTHOR), Hanson, Shawn² (AUTHOR), Juarez, Rucha² (AUTHOR), Wilson, Sean² (AUTHOR), Schroeder, Hobe² (AUTHOR), Li, Qian³ (AUTHOR), Zhu, Lingchao⁴ (AUTHOR), Zhang, Guangyu⁵ (AUTHOR), Blood, Arlin B.^1,2 (AUTHOR) ablood@llu.edu

Publikováno v: Advanced Science. 7/10/2024, Vol. 11 Issue 26, p1-14. 14p.

Investigation of the Dysfunction Caused by High Glucose, Advanced Glycation End Products, and Interleukin-1 Beta and the Effects of Therapeutic Agents on the Microphysiological Artery Model.

Autor: Nam U; Department of Mechanical Engineering, Korea Advanced Institute of Science and Technology (KAIST), Daejeon, 34141, Republic of Korea.; Center for Scientific Instrumentation, Korea Basic Science Institute (KBSI), Daejeon, 34133, Republic of Korea., Kim J; Department of Mechanical Engineering, Korea Advanced Institute of Science and Technology (KAIST), Daejeon, 34141, Republic of Korea., Yi HG; Department of Convergence Biosystems Engineering, Chonnam National University, Gwangju, 61186, Republic of Korea.; Interdisciplinary Program in IT-Bio Convergence System, Chonnam National University, Gwangju, 61186, Republic of Korea., Jeon JS; Department of Mechanical Engineering, Korea Advanced Institute of Science and Technology (KAIST), Daejeon, 34141, Republic of Korea.

Publikováno v: Advanced healthcare materials [Adv Healthc Mater] 2024 Aug; Vol. 13 (21), pp. e2302682. Date of Electronic Publication: 2024 Apr 15.

Inflammatory mediators induce sequestration of VE-cadherin in cultured human endothelial cells

Autor: J S, Alexander, B C, Alexander, L A, Eppihimer, N, Goodyear, R, Haque, C P, Davis, T J, Kalogeris, D L, Carden, Y N, Zhu, C G, Kevil

Publikováno v: Inflammation. 24(2)

The mechanisms through which inflammatory mediators modify endothelial junctional structure are not well understood. Endothelial cells exposed to 1 mM H2O2, 0.1 mM histamine or 4 mM EDTA displayed decreased amounts of VE-cadherin on the cell surface

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::7cfd45bb05f1d0c65f349f7be27773f7
https://pubmed.ncbi.nlm.nih.gov/10718113