Zobrazeno 1 - 10
of 67
pro vyhledávání: '"Byers, LW"'
Publikováno v:
Blood Pressure. 2:183-188
Previously we reported a case of persistent hypotension associated with hypermedullipinemia (Blood Pressure 1992; 1:138-148). The hypermedullipinemia appeared to result from the autonomous secretion of medullipin I (Med I) by renomedullary interstiti
Publikováno v:
The American Journal of the Medical Sciences. 303:86-89
Vasodepressor prostaglandins (PGs), PGE 2 , PGI 2 , and medullipin I (Med I) are synthesized in the kidney. These vasodilator substances are thought to be involved in the antihypertensive function of the kidney. At issue is whether there are biologic
Publikováno v:
Blood Pressure. 1:138-148
A new syndrome is described in a patient with advanced renal insufficiency. This consists of severe and persistent hypotension causing weakness but associated with a clear mental status. Also present is evidence for decreased vascular reactivity. The
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 18:409-417
SUMMARY 1. Medullipin I (Med I) is a hormone extracted from renal papillae and its renomedullary interstitial cells (RIC). Med I is stimulated by elevation of the renal artery perfusion pressure. 2. When isolated normal rat kidneys were perfused eith
Publikováno v:
Clinical Science. 51:287s-290s
1. Allogenic transplants of cultured renomedullary interstitial cells exert a powerful anti-hypertensive action. The blood pressure of hypertensive animals usually drops slowly over 8–12 h whereas the pulse is unchanged or reduced. 2. Lipids derive
Publikováno v:
Blood. 59:582-585
The effect of pure synthetic PAF (1–0-alkyl-2-acetyl-sn-glycero-3- phosphorylcholine) was studied in human platelets. PAF (0.2--2.0 micrograms/ml) produced a dose-dependent aggregation in human platelet- rich plasma (PRP) or platelet suspension obt
Publikováno v:
Blood; March 1982, Vol. 59 Issue: 3 p582-585, 4p
Publikováno v:
The American journal of the medical sciences. 298(2)
Medullipin I causes a delayed onset depressor response when injected intravenously into rats. The glyceryl compounds selachyl alcohol (SA) and monoolein (MO) cause similar vasodepression. The neutral lipid 1-O-hexadecyl-2-acetyl-sn-glycerol (HAG) was
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