Zobrazeno 1 - 10
of 1 252
pro vyhledávání: '"Busuttil RW"'
Autor:
Venick, RS *, McDiarmid, SV, Farmer, DG, Gornbein, J, Martin, MG, Vargas, JH, Ament, ME, Busuttil, RW
Publikováno v:
In American Journal of Transplantation April 2007 7(4):955-963
Autor:
Zhang, C, Zhang, Y, Liu, Y, Kageyama, S, Shen, X-D, Gao, F, Zheng, S, Busuttil, RW, Shaw, GD, Ji, H, Kupiec-Weglinski, JW
Publikováno v:
American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons, vol 17, iss 6
Liver endothelial cell (LEC) damage is essential in the pathogenesis of ischemia-reperfusion injury (IRI) in transplant recipients. We analyzed the mechanism of LEC resistance against IRI by using a novel recombinant soluble form of P selectin glycop
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::1c58c47c2b679650c7e95d55e4a70c9a
https://escholarship.org/uc/item/09s5d5hr
https://escholarship.org/uc/item/09s5d5hr
Autor:
Zarrinpar, A, Busuttil, RW
Publikováno v:
Zarrinpar, A; & Busuttil, RW. (2015). Liver transplantation: Evading antigens-ABO-incompatible liver transplantation. Nature Reviews Gastroenterology and Hepatology, 12(12), 676-677. doi: 10.1038/nrgastro.2015.193. UCLA: Retrieved from: http://www.escholarship.org/uc/item/6848w10p
© 2015 Macmillan Publishers Limited. All rights reserved. Whereas the status of the liver as privileged and exempt from humoral rejection has fallen, attempts at breaching the blood group barrier during transplantation have continued. Previously res
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od_______325::ef25c2cc8463e85a292445a947be783a
http://www.escholarship.org/uc/item/6848w10p
http://www.escholarship.org/uc/item/6848w10p
Publikováno v:
American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons, vol 15, iss 11
Yue, S; Zhou, HM; Zhu, JJ; Rao, JH; Busuttil, RW; Kupiec-Weglinski, JW; et al.(2015). Hyperglycemia and liver ischemia reperfusion injury: A role for the advanced glycation endproduct and its receptor pathway. American Journal of Transplantation, 15(11), 2877-2887. doi: 10.1111/ajt.13360. UCLA: Retrieved from: http://www.escholarship.org/uc/item/7696q1r4
Yue, S; Zhou, HM; Zhu, JJ; Rao, JH; Busuttil, RW; Kupiec-Weglinski, JW; et al.(2015). Hyperglycemia and liver ischemia reperfusion injury: A role for the advanced glycation endproduct and its receptor pathway. American Journal of Transplantation, 15(11), 2877-2887. doi: 10.1111/ajt.13360. UCLA: Retrieved from: http://www.escholarship.org/uc/item/7696q1r4
© Copyright 2015 The American Society of Transplantation and the American Society of Transplant Surgeons. Although pretransplant diabetes is a risk factor for mortality post-liver transplant, the underlying mechanism has not been fully defined. In a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::9f826f31464f82df107665ab4ad09ce5
https://escholarship.org/uc/item/7696q1r4
https://escholarship.org/uc/item/7696q1r4
Publikováno v:
Hepatology (Baltimore, Md.), vol 60, iss 6
Ji, H; Liu, Y; Zhang, Y; Shen, XD; Gao, F; Busuttil, RW; et al.(2014). T-cell immunoglobulin and mucin domain 4 (TIM-4) signaling in innate immune-mediated liver ischemia-reperfusion injury. Hepatology, 60(6), 2052-2064. doi: 10.1002/hep.27334. UCLA: Retrieved from: http://www.escholarship.org/uc/item/7c060134
Ji, H; Liu, Y; Zhang, Y; Shen, XD; Gao, F; Busuttil, RW; et al.(2014). T-cell immunoglobulin and mucin domain 4 (TIM-4) signaling in innate immune-mediated liver ischemia-reperfusion injury. Hepatology, 60(6), 2052-2064. doi: 10.1002/hep.27334. UCLA: Retrieved from: http://www.escholarship.org/uc/item/7c060134
© 2014 by the American Association for the Study of Liver Diseases. Hepatic ischemia-reperfusion injury (IRI), an innate immunity-driven inflammation response, occurs in multiple clinical settings including liver resection, transplantation, trauma,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::89e3f7adc8947aaf9a7821e496451e2c
https://escholarship.org/uc/item/7c060134
https://escholarship.org/uc/item/7c060134
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950), vol 192, iss 11
Yue, S; Rao, J; Zhu, J; Busuttil, RW; Kupiec-Weglinski, JW; Lu, L; et al.(2014). Myeloid PTEN deficiency protects livers from ischemia reperfusion injury by facilitating M2 macrophage differentiation. Journal of Immunology, 192(11), 5343-5353. doi: 10.4049/jimmunol.1400280. UCLA: Retrieved from: http://www.escholarship.org/uc/item/9n81w0xm
Yue, S; Rao, J; Zhu, J; Busuttil, RW; Kupiec-Weglinski, JW; Lu, L; et al.(2014). Myeloid PTEN deficiency protects livers from ischemia reperfusion injury by facilitating M2 macrophage differentiation. Journal of Immunology, 192(11), 5343-5353. doi: 10.4049/jimmunol.1400280. UCLA: Retrieved from: http://www.escholarship.org/uc/item/9n81w0xm
Although the role of phosphatase and tensin homolog deleted on chromosome 10 (PTEN) in regulating cell proliferation is well established, its function in immune responses remains to be fully appreciated. In the current study, we analyzed myeloidspeci
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::4c82e0b101b386368f9535a28e03a570
https://escholarship.org/uc/item/9n81w0xm
https://escholarship.org/uc/item/9n81w0xm
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