Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Bun Seng Tea"'
Publikováno v:
Journal of Vascular Research. 38:462-470
We previously observed that nifedipine reduces aortic hypertrophy in spontaneously hypertensive rats (SHR) partly by inducing smooth muscle cell (SMC) apoptosis. The present study examined nifedipine regulation of SMC apoptosis in carotids with or wi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::93c17398a9e60f5ee1bccb6fe177395c
https://doi.org/10.1159/000051079
https://doi.org/10.1159/000051079
Autor:
T.-V. Dam, Bun-Seng Tea, Pavel Hamet, Denis deBlois, Sergei N. Orlov, Emmanuel Teiger, Lucie Richard, Johanne Tremblay
Publikováno v:
Canadian Journal of Physiology and Pharmacology. 74:850-861
Vascular structure plays a key role in the pathogenesis of hypertension. Because it is less readily reversible than protein accumulation, DNA accumulation in the arterial wall may be considered as a record of past episodes of vascular growth, contrib
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4659c466164a4f8114e5b3663cadfdeb
https://doi.org/10.1139/y96-076
https://doi.org/10.1139/y96-076
Autor:
Louis Gaboury, Lucie Richard, Emmanuel Teiger, C Wisnewsky, Bun-Seng Tea, Ketty Schwartz, Johanne Tremblay, V D Than, Pavel Hamet
Publikováno v:
Journal of Clinical Investigation. 97:2891-2897
Pressure overload induces cardiac growth in the rat, which implies the hypertrophy of cardiac muscle cells and proliferation of nonmuscle cells. The cardiac cell loss observed in parallel has generally been attributed to necrosis. Using an in situ as
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::73b2293b5e2b595dbe18a28fbb5f2dc9
https://doi.org/10.1172/jci118747
https://doi.org/10.1172/jci118747
Publikováno v:
Journal of the American Society of Hypertension : JASH. 7(2)
We previously reported that the AT1 receptor antagonist valsartan and the angiotensin converting enzyme (ACE) inhibitor enalapril decrease DNA synthesis and stimulate apoptosis in interstitial fibroblasts and epicardial mesothelial cells during regre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0dd98f7b8f2a961cea00bd75d0952b0f
https://pubmed.ncbi.nlm.nih.gov/23414835
https://pubmed.ncbi.nlm.nih.gov/23414835
Publikováno v:
Canadian journal of physiology and pharmacology. 83(1)
Cell growth and survival are potential therapeutic targets for the control of complications associated with hypertension. In most cardiovascular disorders, cardiac fibroblasts and large-vessel smooth muscle cells can replicate and thus contribute to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::01c0032c8e4143dcdab5c4bbafbb7d98
https://pubmed.ncbi.nlm.nih.gov/15759048
https://pubmed.ncbi.nlm.nih.gov/15759048
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 35(5)
Abstract —Angiotensin type 2 (AT 2 ) receptors for angiotensin II suppress cell growth and induce apoptosis in vitro, but their role is poorly defined in vivo. We reported that transient induction of smooth muscle cell (SMC) apoptosis precedes DNA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::73feb983092f956ed98dccafd181f1b4
https://pubmed.ncbi.nlm.nih.gov/10818066
https://pubmed.ncbi.nlm.nih.gov/10818066
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 30(3 Pt 2)
Abstract Apoptosis is involved in neonatal remodeling of organs of the cardiovascular system. Since we previously reported hyperplasia of these organs at birth in several forms of genetic hypertension, the aim of this study was to determine whether a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::abde27308f56aa298696430562179e01
https://pubmed.ncbi.nlm.nih.gov/9323012
https://pubmed.ncbi.nlm.nih.gov/9323012
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 29(1 Pt 2)
We previously reported that apoptosis is increased in smooth muscle cells cultured from the aorta of spontaneously hypertensive rats versus normotensive controls. As an initial in vivo exploration, we now examined smooth muscle cell apoptosis regulat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ad327e90a58498d6860b7a949fc0cdf6
https://pubmed.ncbi.nlm.nih.gov/9039125
https://pubmed.ncbi.nlm.nih.gov/9039125
Autor:
Ulrich Pohl, Enming J. Su, Ulrich Hoffmann, Yves-Jacques Schneider, John H. Tinsley, Marc Husmann, Fabienne Kinard, Kathy Jaworski, Brent J.F. Hill, Todd J. Anderson, Elena E. Ustinova, Axel R. Pries, Anders J. Leu, Georg Gradl, Sarah J George, Matthias Schürmann, Mack H. Wu, Susan C. Stevenson, Bun Seng Tea, Brian R. Wamhoff, Daniel J. Goldstein, Johannes Zaspel, Gene Liau, Paul Holvoet, David C. Zawieja, Thérèse Sergent-Engelen, Wenjuan Xu, Pavel Hamet, Claude Remacle, Hans-Anton Lehr, Jennifer Marshall-Neff, Michele Rollence, Frank Christ, Subodh Verma, Kieren J. Mather, Alexander Wipfel, U.K. Franzeck, Christopher L. Jackson, Sarah Y. Yuan, Melanie A. Smith, Paul P. Van Veldhoven, Jacinthe Lemay, Roberto Frisullo, Jason L. Johnson, Torsten Held, Denis deBlois, André Trouet, Michael Sturek
Publikováno v:
Journal of Vascular Research. 39:89-89
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::159b12ec68f9725e002651077ee8a2cf
https://doi.org/10.1159/000048998
https://doi.org/10.1159/000048998
Autor:
Lemay, Jacinthe1, Bun Seng Tea, Jacinthe1,2, Hamet, Pavel2, deBlois, Denis1 denis.deblois@umontreal.ca
Publikováno v:
Journal of Vascular Research. 2001, Vol. 38 Issue 5, p462-470. 9p. 1 Color Photograph, 4 Charts, 1 Graph.