Zobrazeno 1 - 10 of 17 pro vyhledávání: '"Bullen JW"'
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4
Akademický článek
Protein kinase A-dependent phosphorylation stimulates the transcriptional activity of hypoxia-inducible factor 1.
Autor: Bullen JW; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA., Tchernyshyov I; Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA., Holewinski RJ; Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA., DeVine L; Department of Biological Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA., Wu F; Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA., Venkatraman V; Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA., Kass DL; Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA., Cole RN; Department of Biological Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA., Van Eyk J; Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA., Semenza GL; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. Department of Biological Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. Department of Pediatrics, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. Department of Radiation Oncology, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. gsemenza@jhmi.edu.
Publikováno v: Science signaling [Sci Signal] 2016 May 31; Vol. 9 (430), pp. ra56. Date of Electronic Publication: 2016 May 31.
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5
Akademický článek
Hypoxia induces the breast cancer stem cell phenotype by HIF-dependent and ALKBH5-mediated m⁶A-demethylation of NANOG mRNA.
Autor: Zhang C; Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205; Organ Transplant Center, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou 510080, China;, Samanta D; Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Lu H; Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Bullen JW; Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Zhang H; Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Chen I; Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205;, He X; Organ Transplant Center, The First Affiliated Hospital, Sun Yat-sen University, Guangzhou 510080, China;, Semenza GL; Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205; Department of Pediatrics, Johns Hopkins University School of Medicine, Baltimore, MD 21205; Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205; Department of Oncology, Johns Hopkins University School of Medicine, Baltimore, MD 21205; Department of Radiation Oncology, Johns Hopkins University School of Medicine, Baltimore, MD 21205; Department of Biological Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD 21205 gsemenza@jhmi.edu.
Publikováno v: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2016 Apr 05; Vol. 113 (14), pp. E2047-56. Date of Electronic Publication: 2016 Mar 21.
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6
Akademický článek
HIF-1 regulates CD47 expression in breast cancer cells to promote evasion of phagocytosis and maintenance of cancer stem cells.
Autor: Zhang H; Department of Breast Surgery, The First Affiliated Hospital of Xi'an Jiaotong University Health Science Center, Xi'an, Shaanxi 710061, China; Institute for Cell Engineering, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Lu H; Institute for Cell Engineering, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Xiang L; Institute for Cell Engineering, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Bullen JW; Institute for Cell Engineering, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Zhang C; Institute for Cell Engineering, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Samanta D; Institute for Cell Engineering, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Gilkes DM; Department of Oncology, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, He J; Department of Breast Surgery, The First Affiliated Hospital of Xi'an Jiaotong University Health Science Center, Xi'an, Shaanxi 710061, China;, Semenza GL; Institute for Cell Engineering, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; Department of Oncology, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; Department of Pediatrics, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; Department of Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; Department of Radiation Oncology, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; Department of Biological Chemistry, The Johns Hopkins University School of Medicine, Baltimore, MD 21205 gsemenza@jhmi.edu.
Publikováno v: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2015 Nov 10; Vol. 112 (45), pp. E6215-23. Date of Electronic Publication: 2015 Oct 28.
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7
Akademický článek
Chemotherapy triggers HIF-1-dependent glutathione synthesis and copper chelation that induces the breast cancer stem cell phenotype.
Autor: Lu H; Johns Hopkins Institute for Cell Engineering and McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Samanta D; Johns Hopkins Institute for Cell Engineering and McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Xiang L; Johns Hopkins Institute for Cell Engineering and McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Zhang H; Johns Hopkins Institute for Cell Engineering and McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Hu H; Johns Hopkins Institute for Cell Engineering and McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Chen I; Johns Hopkins Institute for Cell Engineering and McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Bullen JW; Johns Hopkins Institute for Cell Engineering and McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205;, Semenza GL; Johns Hopkins Institute for Cell Engineering and McKusick-Nathans Institute of Genetic Medicine, The Johns Hopkins University School of Medicine, Baltimore, MD 21205; Departments of Pediatrics, Medicine, Oncology, Radiation Oncology, and Biological Chemistry, The Johns Hopkins University School of Medicine, Baltimore, MD 21205 gsemenza@jhmi.edu.
Publikováno v: Proceedings of the National Academy of Sciences of the United States of America [Proc Natl Acad Sci U S A] 2015 Aug 18; Vol. 112 (33), pp. E4600-9. Date of Electronic Publication: 2015 Jul 30.
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8
Akademický článek
HIF-1α and TAZ serve as reciprocal co-activators in human breast cancer cells.
Autor: Xiang L; Department of Oncology and Southwest Cancer Center, Southwest Hospital, Third Military Medical University, Chongqing, China.; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD, USA.; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD, USA., Gilkes DM; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD, USA.; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD, USA., Hu H; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD, USA.; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD, USA., Luo W; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD, USA.; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD, USA., Bullen JW; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD, USA.; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD, USA., Liang H; Department of Oncology and Southwest Cancer Center, Southwest Hospital, Third Military Medical University, Chongqing, China., Semenza GL; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD, USA.; McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD, USA.; Departments of Pediatrics, Medicine, Oncology, Radiation Oncology, and Biological Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD, USA.
Publikováno v: Oncotarget [Oncotarget] 2015 May 20; Vol. 6 (14), pp. 11768-78.
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9
Akademický článek
Hypoxia-inducible factor 1 mediates TAZ expression and nuclear localization to induce the breast cancer stem cell phenotype.
Autor: Xiang L; Department of Oncology and Southwest Cancer Center, Southwest Hospital, Third Military Medical University, Chongqing, China. Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD., Gilkes DM; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD., Hu H; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD., Takano N; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD. Department of Biochemistry, School of Medicine, Keio University, Tokyo, Japan., Luo W; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD., Lu H; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD., Bullen JW; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD., Samanta D; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD., Liang H; Department of Oncology and Southwest Cancer Center, Southwest Hospital, Third Military Medical University, Chongqing, China., Semenza GL; Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD. McKusick-Nathans Institute of Genetic Medicine, Johns Hopkins University School of Medicine, Baltimore, MD. Departments of Pediatrics, Medicine, Oncology, Radiation Oncology, and Biological Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD.
Publikováno v: Oncotarget [Oncotarget] 2014 Dec 30; Vol. 5 (24), pp. 12509-27.
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10
Akademický článek
Cross-talk between two essential nutrient-sensitive enzymes: O-GlcNAc transferase (OGT) and AMP-activated protein kinase (AMPK).
Autor: Bullen JW; Department of Biological Chemistry, The Johns Hopkins University School of Medicine, Baltimore, Maryland 21205., Balsbaugh JL; Department of Chemistry, University of Virginia, Charlottesville, Virginia 22904., Chanda D; Department of Molecular Genetics, Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, 6200 MD Maastricht, The Netherlands., Shabanowitz J; Department of Chemistry, University of Virginia, Charlottesville, Virginia 22904., Hunt DF; Department of Chemistry, University of Virginia, Charlottesville, Virginia 22904; Department of Pathology, University of Virginia, Charlottesville, Virginia 22904., Neumann D; Department of Molecular Genetics, Cardiovascular Research Institute Maastricht (CARIM), Maastricht University, 6200 MD Maastricht, The Netherlands., Hart GW; Department of Biological Chemistry, The Johns Hopkins University School of Medicine, Baltimore, Maryland 21205. Electronic address: gwhart@jhmi.edu.
Publikováno v: The Journal of biological chemistry [J Biol Chem] 2014 Apr 11; Vol. 289 (15), pp. 10592-10606. Date of Electronic Publication: 2014 Feb 21.
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