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Mitochondria function associated genes contribute to Parkinson’s disease risk and later age at onset
Autor:
Billingsley, K. J. (Kimberley J.), Barbosa, I. A. (Ines A.), Bandrés-Ciga, S. (Sara), Quinn, J. P. (John P.), Bubb, V. J. (Vivien J.), Deshpande, C. (Charu), Botia, J. A. (Juan A.), Reynolds, R. H. (Regina H.), Zhang, D. (David), Simpson, M. A. (Michael A.), Blauwendraat, C. (Cornelis), Gan-Or, Z. (Ziv), Raphael Gibbs, J. (J.), Nalls, M. A. (Mike A.), Singleton, A. (Andrew), Ryten, M. (Mina), Koks, S. (Sulev)
Mitochondrial dysfunction has been implicated in the etiology of monogenic Parkinson’s disease (PD). Yet the role that mitochondrial processes play in the most common form of the disease; sporadic PD, is yet to be fully established. Here, we compre
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=od______2423::69eee879ee92fbb09575b2a451774566
http://urn.fi/urn:nbn:fi-fe2019092629852
http://urn.fi/urn:nbn:fi-fe2019092629852
