Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Bruce Dangerfield"'
Autor:
Vilasini Shetty, Fabiana Nascimben, Poluru L. Reddy, Azra Raza, Leena Joshi, Naomi Galili, Sairah Alvi, Bruce Dangerfield, Krishnan Allampallam, Ahmed Shaher, Seema Hussaini, Suneel D. Mundle
Publikováno v:
British Journal of Haematology. 116:817-825
Transmission electron microscopic (TEM) studies have not been reported in bone marrow (BM) biopsies of patients with myelodysplastic syndromes (MDS) owing to failure to overcome the technical impediment of maintaining ultrastructural detail in decalc
Autor:
Laurie Lisak, Suneel D. Mundle, Xiao-Ke Huang, Azra Raza, R. Z. Borok, Leena Joshi, Krishnan Allampallam, Bruce Dangerfield, Shalini Anthwal, Poluru L. Reddy, Ahmed Shaher, Sefer Gezer, Francesca Zorat, Leslie Little, Sairah Alvi, Naomi Galili, Benita Henderson, Vilasini Shetty
Publikováno v:
Leukemia Research. 25:941-954
We used bone marrow biopsies instead of mononuclear cells to maintain long-term cultures from 103 patients belonging to all five sub-categories of myelodysplastic syndromes (MDS), as well as 12 normal controls. By week 4, 30-50% confluency was reache
Autor:
Azra Raza, Parameswaran Venugopal, Sairah Alvi, Krishnan Allampallam, Khwaja Aftab Rashid, Bruce Dangerfield, Daniel Zeitler, Evans K. Afenya, Suneel D. Mundle, Jonathan D. Cartlidge, Vilasini Shetty
Publikováno v:
Cancer Letters. 164:197-205
The bone marrow (BM) in myelodysplastic syndromes (MDS) undergoes pathobiological changes that mimic an inflammatory process, and hence, an infectious etiology was suspected in these disorders. In the present report, we examined the bone marrow monon
Autor:
Stephanie A. Gregory, La Tanya Broady-Robinson, Azra Raza, Bruce Dangerfield, B. Yifwayimare Mativi, Parameswaran Venugopal, Harvey D. Preisler, Suneel D. Mundle, Biarou Li, Jonathan D. Cartlidge, Vilasini Shetty
Publikováno v:
British Journal of Haematology. 109:376-381
Myelodysplastic syndromes (MDS) have previously been reported to show competitively high rates of apoptosis and proliferation in the bone marrow (BM). Using a double-labelling technique in the present study, we demonstrated that a significantly high
Autor:
Vilasini, Shetty, Seema, Hussaini, Sairah, Alvi, Leena, Joshi, Ahmed, Shaher, Bruce, Dangerfield, Fabiana, Nascimben, Suneel, Mundle, Krishnan, Allampallam, Poluru, Reddy, Naomi, Galili, Azra, Raza
Publikováno v:
British journal of haematology. 116(4)
Transmission electron microscopic (TEM) studies have not been reported in bone marrow (BM) biopsies of patients with myelodysplastic syndromes (MDS) owing to failure to overcome the technical impediment of maintaining ultrastructural detail in decalc
Autor:
Elias H Touma, Salem Abbes, Andrew G. Clark, Scott M. Williams, Anna Piro, Giuseppe Tagarelli, Jacques Loiselet, Anthi Drousiotou, Sarah A. Tishkoff, Robert Varkonyi, Giovanni Destro-Bisol, Antonio Tagarelli, George Argyropoulos, Bruce Dangerfield, Nelie Cahinhinan, Gérard Lefranc, Mark Stoneking
Publikováno v:
Science (New York, N.Y.) 293 (2001): 455–462.
info:cnr-pdr/source/autori:Tishkoff SA, Varkonyi R, Cahinhinan N, Abbes S, Argyropoulos G, Destro-Bisol G, Drousiotou A, Dangerfield B, Lefranc G, Loiselet J, Piro A, Stoneking M, Tagarelli A, Tagarelli G, Touma EH, Williams SM, Clark AG/titolo:Haplotype diversity and linkage disequilibrium at human G6PD: recent origin of alleles that confer malarial resistance./doi:/rivista:Science (New York, N.Y.)/anno:2001/pagina_da:455/pagina_a:462/intervallo_pagine:455–462/volume:293
info:cnr-pdr/source/autori:Tishkoff SA, Varkonyi R, Cahinhinan N, Abbes S, Argyropoulos G, Destro-Bisol G, Drousiotou A, Dangerfield B, Lefranc G, Loiselet J, Piro A, Stoneking M, Tagarelli A, Tagarelli G, Touma EH, Williams SM, Clark AG/titolo:Haplotype diversity and linkage disequilibrium at human G6PD: recent origin of alleles that confer malarial resistance./doi:/rivista:Science (New York, N.Y.)/anno:2001/pagina_da:455/pagina_a:462/intervallo_pagine:455–462/volume:293
The frequencies of low-activity alleles of glucose-6-phosphate dehydrogenase in humans are highly correlated with the prevalence of malaria. These “deficiency” alleles are thought to provide reduced risk from infection by the Plasmodium parasite