Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Brooke C. Henderson"'
Publikováno v:
International Journal of Biological Sciences, Vol 3, Iss 6, Pp 385-392 (2007)
Elevated oxidative stress has been characterized in numerous disorders including systemic hypertension, arterial stiffness, left ventricular hypertrophy (LVH) and heart failure. The peroxisome proliferator activated receptor gamma (PPARγ) ameliorate
Externí odkaz:
https://doaj.org/article/d5c9f7beb9c54d18bdce2eeda996abf4
Autor:
Corey L. Reynolds, Alexander V. Ovechkin, Utpal Sen, Neetu Tyagi, Ganesh K. Kartha, Brooke C. Henderson, Suresh C. Tyagi, Walter E. Rodriguez, Karni S. Moshal
Publikováno v:
International Journal of Biological Sciences
Elevated oxidative stress has been characterized in numerous disorders including systemic hypertension, arterial stiffness, left ventricular hypertrophy (LVH) and heart failure. The peroxisome proliferator activated receptor gamma (PPARgamma) amelior
Autor:
Irving G. Joshua, Karni S. Moshal, Ganesh K. Kartha, Utpal Sen, Brooke C. Henderson, Neetu Tyagi, Suresh C. Tyagi, Dorothea S. Rosenberger
Publikováno v:
Antioxidants & Redox Signaling. 9:971-978
Glucose-mediated impairment of homocysteine (Hcy) metabolism and decrease in renal clearance contribute to hyperhomocysteinemia (HHcy) in diabetes. The Hcy induces oxidative stress, inversely relates to the expression of peroxisome proliferators acti
Autor:
Neetu Tyagi, Karni S. Moshal, Suresh C. Tyagi, Ganesh K. Kartha, Brooke C. Henderson, Alexander V. Ovechkin
Publikováno v:
European Journal of Heart Failure. 9:450-457
Despite extensive strides in understanding pressure overload induced heart failure, there is very little known about oxidative stress induced matrix metalloproteinase (MMP) activation, collagen degradation and remodeling in pressure overload heart fa
Autor:
Brooke C. Henderson, Suresh C. Tyagi
Publikováno v:
Journal of Molecular and Cellular Cardiology. 41:959-962
Takenaka et al. [Takenaka H, Kihara Y, Iwanaga Y, Onozawa Y, Toyokuni S, Kita T. Angiotensin II, oxidative stress, and extracellular matrix degradation during transition to LV failure in rats with hypertension, J Mol Cell Cardiol, 2006; in press] in
Autor:
Neetu Tyagi, Suresh C. Tyagi, Karni S. Moshal, Mesia Steed, Utpal Sen, Brooke C. Henderson, Alexander V. Ovechkin
Publikováno v:
American Journal of Physiology-Cell Physiology. 290:C883-C891
Homocysteine (Hcy) induces matrix metalloproteinase (MMP)-9 in microvascular endothelial cells (MVECs). We hypothesized that the ERK1/2 signaling pathway is involved in Hcy-mediated MMP-9 expression. In cultured MVECs, Hcy induced activation of ERK,
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 288:H2770-H2777
We examined the hypothesis that oxidants generated nitroso derivatives, activated latent matrix metalloproteinase (MMP), and induced proteinase-activated receptor 1 (PAR-1), leading to disconnection between the endothelium and myocytes. Administratio
Autor:
Utpal Sen, William Gillespie, David Lominadze, Brooke C. Henderson, Mesia Steed, Neetu Tyagi, Suresh C. Tyagi, Karni S. Moshal, Dorothea S. Rosenberger
Publikováno v:
The FASEB Journal. 21
Reverse Redox Remodeling in Pressure Overload Induced Heart Failure in mice treated with ciglitazone
Autor:
Ganesh K. Kartha, Brooke C. Henderson, Karni S. Moshal, Alexander V. Ovechkin, Neetu Tyagi, Suresh C. Tyagi, Corey Reynolds
Publikováno v:
The FASEB Journal. 21
Autor:
Neetu Tyagi, Mahavir Singh, Hong Zhang, Brooke C. Henderson, Dorothea S. Rosenberger, Utpal Sen, Karni S. Moshal, Suresh C. Tyagi
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 291(6)
Hyperhomocysteinemia (HHcy) is associated with atherosclerosis, stroke, and dementia. Hcy causes extracellular matrix remodeling by the activation of matrix metalloproteinase-9 (MMP-9), in part, by inducing redox signaling and modulating the intracel