Human cellular model systems of β-thalassemia enable in-depth analysis of disease phenotype.

Autor: Daniels DE; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK., Ferrer-Vicens I; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK., Hawksworth J; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK., Andrienko TN; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK., Finnie EM; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK., Bretherton NS; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK., Ferguson DCJ; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK., Oliveira ASF; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK., Szeto JA; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK., Wilson MC; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK., Brewin JN; Haematology Department, King's college Hospital NHS Foundation, London, SE5 9RS, UK.; Red Cell Biology Group, Kings College London, London, SE5 9NU, UK., Frayne J; School of Biochemistry, University of Bristol, Bristol, BS8 1TD, UK. Jan.Frayne@Bristol.ac.uk.

Publikováno v: Nature communications [Nat Commun] 2023 Oct 06; Vol. 14 (1), pp. 6260. Date of Electronic Publication: 2023 Oct 06.