Zobrazeno 1 - 10
of 156
pro vyhledávání: '"Boyd L. O'Dell"'
Publikováno v:
Journal of Animal Science. 92:2481-2493
The experimental objective was to determine the relative availability of endogenous Zn in soybean protein (SP) and cooked ground beef (GB) compared with the availability of Zn from ZnCO(3) for young swine. Crossbred pigs (4 trials with total n = 104)
Publikováno v:
Nutritional Neuroscience. 5:19-25
Zinc deprivation results in decreased and cyclic food intake in rats. We determined the response of zinc-deprived rats to neuropeptide Y (NPY). In a preliminary experiment, rats were fed a low (-Zn;1 mg/kg) or adequate zinc diet (+Zn; 100 mg/kg) for
Autor:
Boyd L. O'Dell
Publikováno v:
The Journal of Nutrition. 130:1432S-1436S
The concentration of plasma zinc is the generally accepted index of zinc status. Although low plasma zinc is an essential criterion of deficiency, alone it is inadequate. To supplement this index, we sought to determine the first limiting biochemical
Autor:
Boyd L. O'Dell
Publikováno v:
Nutrition Reviews. 51:307-309
Copper is an essential nutrient for optimal function of the immune system; deficiency results in impairment of both humoral and cell-mediated components. Copper deficiency in rodents results in decreased numbers of CD4+ (helper) and total T cells. Th
Autor:
Ruth S. MacDonald, Jimmy D. Browning, William H. Thornton, Boyd L. O'Dell, Lavonna C. Wollard-Biddle
Publikováno v:
The Journal of Nutrition. 128:1600-1605
Growth failure in zinc-deficient animals is associated with decreased DNA synthesis; zinc deprivation of 3T3 cells, by use of diethylenetrinitrilopentaacetate (DTPA), impairs thymidine incorporation when the cells are stimulated with fetal bovine ser
Publikováno v:
The Journal of Nutritional Biochemistry. 8:346-350
Zinc deficiency in rats impairs platelet aggregation, and this defect is associated with the failure of platelets to take up external calcium. Recent data show that the red cell plasma membrane from zinc-deficient rats contains a lower than normal co
Autor:
Jinming Xia, Boyd L. O'Dell
Publikováno v:
The Journal of Nutritional Biochemistry. 6:661-666
Aggregation of rat platelets, when stimulated by adenosine diphosphate (ADP) or fluoride, is impaired by zinc deficiency, and the defect is associated with a decreased uptake of external Ca2+. Zinc deficiency also impairs the aggregatory response of
Autor:
Boyd L. O'Dell, Jimmy D. Browning
Publikováno v:
The Journal of Nutritional Biochemistry. 6:588-594
Zinc deficiency results in neuropathology affecting both the peripheral and central nervous systems. A previous study showed that decreased calcium uptake by cortical synaptosomes was associated with the peripheral neuropathy in guinea pigs. Deficien
Autor:
Boyd L. O'Dell, Jimmy D. Browning
Publikováno v:
Biological trace element research. 152(1)
Zinc deficiency in 3T3 cells induced by the use of diethylenetriaminepentaacetate (DTPA) has been shown to impair calcium entry associated with failure of proliferation when the cells are stimulated with polypeptide growth factors (GF). These functio
Publikováno v:
The Journal of Nutritional Biochemistry. 5:542-546
Addition of zinc to human platelets increases the binding of phorbol esters, and this was postulated to be the result of protein kinase C translocation. To test this hypothesis, washed rat platelets were treated with either a combination of 20 μmol/