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Autor:
Rachel Dean, Tommaso Cassano, William Davies, Trevor Humby, Simon Trent, Bonnie Veit, Gaurav Bedse, Obah A. Ojarikre
Publikováno v:
Psychoneuroendocrinology
Chromosomal deletions at Xp22.3 appear to influence vulnerability to the neurodevelopmental disorders attention deficit hyperactivity disorder (ADHD) and autism. 39,X(Y*)O mice, which lack the murine orthologue of the Xp22.3 ADHD candidate gene STS (