Zobrazeno 1 - 10
of 18
pro vyhledávání: '"Bochra Tourki"'
Autor:
Theodoros Karampitsakos, Apostolos Galaris, Serafeim Chrysikos, Ourania Papaioannou, Ioannis Vamvakaris, Ilianna Barbayianni, Paraskevi Kanellopoulou, Sofia Grammenoudi, Nektarios Anagnostopoulos, Grigoris Stratakos, Matthaios Katsaras, Fotios Sampsonas, Katerina Dimakou, Effrosyni D. Manali, Spyridon Papiris, Bochra Tourki, Brenda M Juan-Guardela, Petros Bakakos, Demosthenes Bouros, Jose D Herazo-Maya, Vassilis Aidinis, Argyris Tzouvelekis
Publikováno v:
Respiratory Research, Vol 24, Iss 1, Pp 1-13 (2023)
Abstract Background Mediastinal lymph node enlargement is prevalent in patients with idiopathic pulmonary fibrosis (IPF). Studies investigating whether this phenomenon reflects specific immunologic activation are lacking. Methods Programmed cell deat
Externí odkaz:
https://doaj.org/article/8c3755ea9f034d7886df93966efb4f18
Autor:
Bochra Tourki, Vasundhara Kain, Amanda B. Pullen, Paul C. Norris, Nirav Patel, Pankaj Arora, Xavier Leroy, Charles N. Serhan, Ganesh V. Halade
Publikováno v:
Molecular Metabolism, Vol 31, Iss , Pp 138-149 (2020)
Objective: Recently, we observed that the specialized proresolving mediator (SPM) entity resolvin D1 activates lipoxin A4/formyl peptide receptor 2 (ALX/FPR2), which facilitates cardiac healing and persistent inflammation is a hallmark of impaired ca
Externí odkaz:
https://doaj.org/article/db743dad70734920abb663bad30b0a4d
Autor:
Bochra Tourki, Ganesh V. Halade
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 8 (2021)
Heart failure with preserved ejection fraction (HFpEF) is an emerging disease with signs of nonresolving inflammation, endothelial dysfunction, and multiorgan defects. Moreover, based on the clinical signs and symptoms and the rise of the obesity epi
Externí odkaz:
https://doaj.org/article/eb66b29d46e24641b26ef53db0bbb9ba
Publikováno v:
Biomedicine & Pharmacotherapy, Vol 139, Iss , Pp 111574- (2021)
The presistent increase of 12/15 lipoxygenase enzyme activity is correlated with uncontrolled inflammation, leading to organ dysfunction. ML351, a potent 12/15 lipoxygenase (12/15LOX) inhibitor, was reported to reduce infarct size and inflammation in
Externí odkaz:
https://doaj.org/article/43a61aa9114f4f44a24ddbb6b26ea777
Autor:
Bochra Tourki, Anais Dumesnil, Elise Belaidi, Slim Ghrir, Diane Godin-Ribuot, Naziha Marrakchi, Vincent Richard, Paul Mulder, Erij Messadi
Publikováno v:
Toxins, Vol 11, Iss 9, p 524 (2019)
Myocardial infarction (MI) followed by left ventricular (LV) remodeling is the most frequent cause of heart failure. Lebetin 2 (L2), a snake venom-derived natriuretic peptide, exerts cardioprotection during acute myocardial ischemia-reperfusion (IR)
Externí odkaz:
https://doaj.org/article/8b888a831b0a4f099fbea59f021bf008
Autor:
Bochra Tourki, Philippe Matéo, Jessica Morand, Mohamed Elayeb, Diane Godin-Ribuot, Naziha Marrakchi, Elise Belaidi, Erij Messadi
Publikováno v:
PLoS ONE, Vol 11, Iss 9, p e0162632 (2016)
Cardiac ischemia is one of the leading causes of death worldwide. It is now well established that natriuretic peptides can attenuate the development of irreversible ischemic injury during myocardial infarction. Lebetin 2 (L2) is a new discovered pept
Externí odkaz:
https://doaj.org/article/50ed1699d2a24a848bfd7cd5abc3094b
Autor:
Vasundhara Kain, Bochra Tourki, Xavier Leroy, Pankaj Arora, Paul C. Norris, Amanda B. Pullen, Ganesh V. Halade, Charles N. Serhan, Nirav Patel
Publikováno v:
Molecular Metabolism, Vol 31, Iss, Pp 138-149 (2020)
Molecular Metabolism
Molecular Metabolism
Objective Recently, we observed that the specialized proresolving mediator (SPM) entity resolvin D1 activates lipoxin A4/formyl peptide receptor 2 (ALX/FPR2), which facilitates cardiac healing and persistent inflammation is a hallmark of impaired car
Publikováno v:
Circulation. 144
Introduction: Age-related cardiac dysfunction is a prime component of sepsis-induced multi-organ failure in the clinical setting. Arachidonate 5-lipoxygenase (5LOX) pathway and 5LOX-derived lipid mediators are critical in early inflammation and immun
Publikováno v:
Metabolism. 96:22-32
Background After myocardial infarction (MI), delayed progression or reversal of cardiac remodeling is a prime target to limit advanced chronic heart failure (HF). However, the temporal kinetics of lipidomic and systemic metabolic signaling is unclear
Autor:
Ganesh V, Halade, Bochra, Tourki
Publikováno v:
Advances in experimental medicine and biology. 1161
After myocardial infarction, splenic leukocytes direct biosynthesis of specialized pro-resolving mediators (SPMs) that are essential for the resolution of inflammation and tissue repair. In a laboratory environment, after coronary ligation of healthy