Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Bjørg Thiellesen Buch"'
Autor:
Natascha Masselkhi Christensen, Stine Ringholm, Bjørg Thiellesen Buch, Anders Gudiksen, Jens Frey Halling, Henriette Pilegaard
Publikováno v:
Experimental Gerontology, Vol 172, Iss , Pp 112046- (2023)
Aging has been suggested to be associated with changes in oxidative capacity, autophagy, and mitophagy in the liver, but a simultaneous evaluation of these key cellular processes is lacking. Moreover, skeletal muscle transcriptional coactivator perox
Externí odkaz:
https://doaj.org/article/b93741ca177e4f6c8f6262dc824f9397
Autor:
Jørgen F. P. Wojtaszewski, Jens Frey Halling, Rasmus Kjøbsted, Mette Algot Olsen, Bjørg Thiellesen Buch, Anders Gudiksen, Henriette Pilegaard, Stine Ringholm
Publikováno v:
FASEB journal : official publication of the Federation of American Societies for Experimental BiologyREFERENCES. 34(6)
The aim of the study was to investigate the impact of autophagy inhibition on skeletal muscle mitochondrial function and glucose homeostasis in young and aged mice. The transcriptional co-activator PGC-1α regulates muscle oxidative phenotype which h
Autor:
Jacob Nøhr-Meldgaard, Henrik Jessen, Bjørg Thiellesen Buch, Anders Gudiksen, Stine Ringholm, Clara Prats, Natascha Masselkhi Christensen, Jens Frey Halling, P. Darrell Neufer, Henriette Pilegaard
Publikováno v:
American journal of physiology. Endocrinology and metabolism. 317(3)
Impaired mitochondrial function has been implicated in the pathogenesis of age-associated metabolic diseases through regulation of cellular redox balance. Exercise training is known to promote mitochondrial biogenesis in part through induction of the
Autor:
Bjørg Thiellesen Buch, Jens Bangsbo, Ylva Hellsten, Stine Ringholm, Anders Gudiksen, Nina Brandt, Lene Rostgaard Nielsen, Henriette Pilegaard
Publikováno v:
American journal of physiology. Endocrinology and metabolism. 314(1)
PGC-1α has been suggested to regulate exercise training-induced metabolic adaptations and autophagy in skeletal muscle. The factors regulating PGC-1α, however, have not been fully resolved. The aim was to investigate the impact of β-adrenergic sig