Zobrazeno 1 - 10
of 26
pro vyhledávání: '"Bing-Ren Hu"'
Publikováno v:
Neurobiology of Disease, Vol 8, Iss 1, Pp 147-154 (2001)
The serine-threonine kinase Akt1 promotes cell survival through inhibition of apoptosis. One of the potential downstream targets of Akt1 is p70 S6 kinase, p70S6K, an enzyme implicated in the regulation of protein synthesis. In this study, we investig
Externí odkaz:
https://doaj.org/article/2e8e508a57b4415fa002f7d575e1b248
Autor:
Bing-Ren Hu, 胡秉仁
89
Based on the Decimation-in-frequency method, the design of FFT pipeline VLSI architecture is proposed in this work. As compared with traditional parallel architecture, the proposed architecture has the advan-tage of reducing the computational
Based on the Decimation-in-frequency method, the design of FFT pipeline VLSI architecture is proposed in this work. As compared with traditional parallel architecture, the proposed architecture has the advan-tage of reducing the computational
Externí odkaz:
http://ndltd.ncl.edu.tw/handle/80823302262973626608
Publikováno v:
World Journal of Clinical Cases
Background DNA damage is one of the critical contributors to the occurrence and development of some cancers. APEX1 and APEX2 are the most important molecules in the DNA damage, and APEX1 has been identified as a diagnostic and prognostic biomarker in
Autor:
Anthony A. Oliva, Bing Ren Hu, Helen M. Bramlett, Coleen M. Atkins, Shaoyi Chen, W. Dalton Dietrich, Ofelia F. Alonso
Publikováno v:
European Journal of Neuroscience. 26:810-819
Traumatic brain injury (TBI) results in significant hippocampal pathology and hippocampal-dependent memory loss, both of which are alleviated by hypothermia treatment. To elucidate the molecular mechanisms regulated by hypothermia after TBI, rats und
Autor:
Myron D. Ginsberg, Raul Busto, Bo K. Siesjö, Miguel A. Perez-Pinzon, Shorena Janelidze, Chun Li Liu, Bing Ren Hu, Thomas J. Sick
Publikováno v:
Journal of Cerebral Blood Flow & Metabolism. 21:865-875
Two hours of transient focal brain ischemia causes acute neuronal death in the striatal core region and a somewhat more delayed type of neuronal death in neocortex. The objective of the current study was to investigate protein aggregation and neurona
Publikováno v:
Neurobiology of Disease, Vol 8, Iss 1, Pp 127-135 (2001)
The present study was undertaken to investigate whether extracellular signal-regulated kinase (ERK) was involved in mediating hyperglycemia-exaggerated cerebral ischemic damage. Phosphorylation of ERK 1/2 was studied by immunocytochemistry and by Wes
Publikováno v:
Neuroscience Research Communications. 28:181-187
Mitochondrial release of cytochrome c and activation of caspases are considered to be parts of a major cell death pathway in cells of various origins. In order to study if caspases are involved in the events leading to cell death induced by oxidants
Autor:
Hiroyuki Osada, Yoshitoshi Kasuya, Tatsuhiko Sudo, Masumi Maruyama, Bing-Ren Hu, Takashi Shiga
Publikováno v:
Brain Research. 887:350-358
p38 has been implicated to play a critical role in regulating apoptosis in PC12 and cerebellar granule cells, and is inactivated in cultured fetal neurons in response to insulin. Though p38 is activated in microglia after ischemia, the physiological
Publikováno v:
Hippocampus. 10:610-616
A transient interruption in cerebral blood flow can lead to delayed neuronal death in certain vulnerable cell populations several days after blood flow is restored. Among the most vulnerable cell populations in the forebrain are hippocampal CA1 pyram
Publikováno v:
Journal of Neurochemistry. 61:1789-1794
Growth factors stimulate cellular protein synthesis, but the intracellular signaling mechanisms that regulate initiation of mRNA translation in neurons have not been clarified. A rate-limiting step in the initiation of protein synthesis is the format