Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Billie Ketelsen"'
Autor:
Matthew D Johnson, Brandon Stone, Bryan J Thibodeau, Andrew M Baschnagel, Sandra Galoforo, Laura E Fortier, Billie Ketelsen, Samreen Ahmed, Zakiya Kelley, Alaa Hana, Thomas G Wilson, John M Robertson, Robert P Jury, George D Wilson
Publikováno v:
Tumor Biology, Vol 39 (2017)
This study investigated the Trk receptor family as a therapeutic target in pancreatic ductal adenocarcinoma and assessed their prognostic significance. Global gene expression analysis was investigated in prospectively collected pancreatic ductal aden
Externí odkaz:
https://doaj.org/article/a1f8a386a57643afb3a6ebcc0ff8ae92
Autor:
Billie Ketelsen
Publikováno v:
American Society for Clinical Laboratory Science.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::986319ca782edbacfe2107164584068c
https://doi.org/10.29074/ascls.2020002287
https://doi.org/10.29074/ascls.2020002287
Autor:
Billie Ketelsen
Publikováno v:
American Society for Clinical Laboratory Science. :ascls.120.002253
Thrombotic thrombocytopenic purpura (TTP) is a multi-faceted disease for a clinical laboratory with diagnosis data and treatment spread across many different laboratory sections. Encompassing results from hematology, chemistry, molecular, and coagula
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0960300cdcf943ded13588e7b20c0651
https://doi.org/10.29074/ascls.120.002253
https://doi.org/10.29074/ascls.120.002253
Autor:
Billie Ketelsen
Publikováno v:
American Society for Clinical Laboratory Science. :ascls.120.002287
Thrombotic Thrombocytopenia Pupura (TTP) is a disease that is classified by abnormal functioning of the ADAMTS13 protease. ADAMTS13 protease impairment can be caused by genetic mutations at the gene level or through autoantibodies that are formed wit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::537c1a6190b8e43224ef5736738d3bb9
https://doi.org/10.29074/ascls.120.002287
https://doi.org/10.29074/ascls.120.002287