Zobrazeno 1 - 10
of 32
pro vyhledávání: '"Bharati Matta"'
Autor:
Bailey K. Roberts, Dan Iris Li, Carter Somerville, Bharati Matta, Vaishali Jha, Adison Steinke, Zarina Brune, Lionel Blanc, Samuel Z. Soffer, Betsy J. Barnes
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-16 (2024)
Abstract Metastasis is driven by extensive cooperation between a tumor and its microenvironment, resulting in the adaptation of molecular mechanisms that evade the immune system and enable pre-metastatic niche (PMN) formation. Little is known of the
Externí odkaz:
https://doaj.org/article/e814d3e28b7740d3bd10af83711e9bac
Autor:
Bharati Matta, Betsy J Barnes, Jason A Carter, Jenna Battaglia, Carter Somerville, Benjamin D Harris, Margaret LaPan, Gurinder S Atwal
Publikováno v:
Journal for ImmunoTherapy of Cancer, Vol 11, Iss 12 (2023)
Background Cancer–testis (CT) genes are targets for tumor antigen-specific immunotherapy given that their expression is normally restricted to the immune-privileged testis in healthy individuals with aberrant expression in tumor tissues. While they
Externí odkaz:
https://doaj.org/article/2cd8f9e8521e45f0b5a7fa181086f738
Publikováno v:
Bio-Protocol, Vol 13, Iss 12 (2023)
Neutrophil extracellular traps (NETs) are web-like structures made up of decondensed chromatin fibers along with neutrophil granular proteins that are extruded by neutrophils after activation or in response to foreign microorganisms. NETs have been a
Externí odkaz:
https://doaj.org/article/b1a46525edf84125bd304be22ba6498f
Publikováno v:
Frontiers in Immunology, Vol 13 (2022)
Neutrophil extracellular traps (NETs) are web-like structures extruded by neutrophils after activation or in response to microorganisms. These extracellular structures are decondensed chromatin fibers loaded with antimicrobial granular proteins, pept
Externí odkaz:
https://doaj.org/article/4a6bc49be0ed4b4d85e0b7c363513bc5
Publikováno v:
Frontiers in Immunology, Vol 9 (2018)
The interferon regulatory factors (IRFs) are a family of master transcription factors that regulate pathogen-induced innate and acquired immune responses. Aberration(s) in IRF signaling pathways due to infection, genetic predisposition and/or mutatio
Externí odkaz:
https://doaj.org/article/3f4c28b7d88e4b11b1e801f59c423104
Autor:
Jason A. Carter, Bharati Matta, Jenna Battaglia, Carter Somerville, Benjamin D. Harris, Margaret LaPan, Gurinder S. Atwal, Betsy J. Barnes
Publikováno v:
bioRxiv
BackgroundCancer-testis (CT) genes are targets for tumor antigen-specific immunotherapy given that their expression is normally restricted to the immune-privileged testis in healthy individuals with aberrant expression in tumor tissues. While they re
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d1655f59289b5c222a22c6256a350f4c
https://doi.org/10.1101/2023.05.09.539617
https://doi.org/10.1101/2023.05.09.539617
Publikováno v:
Graefe's Archive for Clinical and Experimental Ophthalmology. 257:953-960
Experimental autoimmune anterior uveitis (EAAU) is a clinically relevant animal model for human idiopathic anterior uveitis (IAU). The role of the immunomodulator transforming growth factor β2 (TGF-β2) in EAAU pathology is unknown. In this study, w
Publikováno v:
The Journal of Immunology. 208:158.01-158.01
SLE is a complex multifactorial autoimmune disease characterized by high levels of autoantibodies that impact many organs. Neutrophil extracellular traps (NETs) are a potential source of antigen leading to the production of autoantibodies, as antibod
Publikováno v:
The Journal of Immunology. 208:105.32-105.32
The recruitment of leukocytes to sites of infection is an essential component of the innate immune response to pathogens. Defects in leukocyte migration can render the host susceptible to recurrent infections. In our efforts to identify new mediators
Publikováno v:
The Journal of Immunology. 208:165.02-165.02
Systemic Lupus Erythematosus (SLE) is a devastating autoimmune disease that results from failure of the immune system to distinguish “self” from “non-self”. Studies in our lab and others demonstrated that human SLE CD4+ T cells have elevated