Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Bettina Nordbo"'
Autor:
Brian R. Anderson, Marianne L. Jensen, Peter H. Hagedorn, Sean C. Little, Richard E. Olson, Ron Ammar, Bernadette Kienzle, John Thompson, Ivar McDonald, Stephen Mercer, Jonas Vikesaa, Bettina Nordbo, Larry Iben, Yang Cao, Joanne Natale, Greg Dalton-Kay, Angela Cacace, Bo R. Hansen, Maj Hedtjärn, Troels Koch, Linda J. Bristow
Publikováno v:
Molecular Therapy: Nucleic Acids, Vol 19, Iss , Pp 1290-1298 (2020)
Hundreds of dominant-negative myosin mutations have been identified that lead to hypertrophic cardiomyopathy, and the biomechanical link between mutation and disease is heterogeneous across this patient population. To increase the therapeutic feasibi
Externí odkaz:
https://doaj.org/article/ebaf673f331045b1903490834433fa8c
Autor:
Filippo Sladojevich, Remo Gamboni, Tanja Minz, Philippe Ringler, Rainer Alex, Sabine Kux van Geijtenbeek, Michaela Portmann, Axel Ducret, Matthias E. Lauer, Ravi Jagasia, Eric-André Kusznir, Bettina Nordbo, Sabine Sewing, Michael Keller, Nikhil J. Pandya, Erich Koller, Philip Grossen, Marco Berrera, Martina Brigitte Duschmalé, Sylwia Huber, Marianne Lerbech Jensen
Publikováno v:
European Journal of Pharmaceutics and Biopharmaceutics. 158:198-210
The natural capacity of extracellular vesicles (EVs) to transport their payload to recipient cells has raised big interest to repurpose EVs as delivery vehicles for xenobiotics. In the present study, bovine milk-derived EVs (BMEVs) were investigated
Autor:
Bo Hansen, Brian R. Anderson, Linda J. Bristow, Jonas Vikesaa, Sean C. Little, JoAnne E Natale, Maj Hedtjarn, Greg Dalton-Kay, John Ryan Thompson, Yang Cao, Peter Hagedorn, Marianne Lerbech Jensen, Ivar M. McDonald, Bernadette Kienzle, Larry Iben, Stephen E. Mercer, Bettina Nordbo, Angela Cacace, Troels Koch, Richard E. Olson, Ron Ammar
Publikováno v:
Molecular Therapy: Nucleic Acids, Vol 19, Iss, Pp 1290-1298 (2020)
Molecular Therapy. Nucleic Acids
Molecular Therapy. Nucleic Acids
Hundreds of dominant-negative myosin mutations have been identified that lead to hypertrophic cardiomyopathy, and the biomechanical link between mutation and disease is heterogeneous across this patient population. To increase the therapeutic feasibi