Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Bethany T. Chan"'
Autor:
Nicola Steel, Aduragbemi A. Faniyi, Sayema Rahman, Stefanie Swietlik, Beata I. Czajkowska, Bethany T. Chan, Alexander Hardgrave, Anthony Steel, Tim D. Sparwasser, Mushref B. Assas, Richard K. Grencis, Mark A. Travis, John J. Worthington
Publikováno v:
PLoS Pathogens, Vol 17, Iss 11 (2021)
Externí odkaz:
https://doaj.org/article/404f53da9a8945efbdf3d5ee272feda3
Autor:
Nicola Steel, Aduragbemi A Faniyi, Sayema Rahman, Stefanie Swietlik, Beata I Czajkowska, Bethany T Chan, Alexander Hardgrave, Anthony Steel, Tim D Sparwasser, Mushref B Assas, Richard K Grencis, Mark A Travis, John J Worthington
Publikováno v:
PLoS Pathogens, Vol 15, Iss 4, p e1007657 (2019)
Helminths are highly prevalent metazoan parasites that infect over a billion of the world's population. Hosts have evolved numerous mechanisms to drive the expulsion of these parasites via Th2-driven immunity, but these responses must be tightly cont
Externí odkaz:
https://doaj.org/article/4df84396880e471badbb38cffcabcad0
Autor:
Alexander Hardgrave, Richard K. Grencis, Tim Sparwasser, Aduragbemi A Faniyi, Anthony Steel, Beata I. Czajkowska, John J. Worthington, Bethany T. Chan, Stefanie Swietlik, Nicola Steel, Mushref Bakri Assas, Mark A. Travis, Sayema Rahman
Publikováno v:
Steel, N, Faniyi, A A, Rahman, S, Swietlik, S, Czajkowska, B, Chan, B T, Hardgrave, A, Steel, A, Sparwasser, T, Assas, M, Grencis, R, Travis, M & Worthington, J J 2019, ' TGFβ-activation by dendritic cells drives Th17 induction and intestinal contractility and augments the expulsion of the parasite Trichinella spiralis in mice ', PL o S Pathogens . https://doi.org/10.1371/journal.ppat.1007657
PLoS Pathogens
PLoS Pathogens, Vol 15, Iss 4, p e1007657 (2019)
PLoS pathogens
PLoS Pathogens
PLoS Pathogens, Vol 15, Iss 4, p e1007657 (2019)
PLoS pathogens
Helminths are highly prevalent metazoan parasites that infect over a billion of the world’s population. Hosts have evolved numerous mechanisms to drive the expulsion of these parasites via Th2-driven immunity, but these responses must be tightly co