Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Bernd Matiba"'
Publikováno v:
Journal of Immunological Methods. 193:63-70
CD95L (CD95/APO-1/Fas ligand) is a type II transmembrane glycoprotein that induces apoptosis in sensitive target cells. CD95L can be proteolytically cleaved from the membrane by a metalloprotease and occurs in a soluble form. Thus CD95L may act as a
Autor:
Renata Flury, Patrick Aebischer, Michael Weller, Karl Frei, Peter H. Krammer, Sara M. Mariani, Bernd Matiba, Anne Rensing-Ehl, Adriano Fontana
Publikováno v:
European Journal of Immunology. 25:2253-2258
Fas/APO-1 (CD95) is a cell surface receptor which mediates apoptosis when ligated by specific antibodies or by its recently cloned natural ligand, FasL. We have studied the cytotoxic potential of FasL in vivo using Fas/APO-1-expressing Yac-1 cells as
Publikováno v:
The European journal of health economics : HEPAC : health economics in prevention and care. 7(4)
Obesity is associated with major health risks and a high economic burden impacting on health care systems. This study utilises the latest evidence from randomised clinical trials (RCTs) to explore and to assess the cost effectiveness of sibutramine i
Autor:
Bernd, Matiba
Publikováno v:
MMW Fortschritte der Medizin. 144(27-28)
Publikováno v:
European journal of immunology. 25(10)
The CD95 (APO-1/Fas) ligand (CD95L) mediates apoptosis in sensitive target cells, Ca(2+)-independent cytotoxicity of cells from perforin knock-out mice, and peripheral deletion of activated T cells through engagement of its cognate receptor CD95. Dou
Publikováno v:
European journal of immunology. 25(8)
APO-1/Fas (CD95) ligand (APO-1L) induces apoptosis in sensitive target cells. Activation-induced T cell death and Ca2(+)-independent cytotoxicity in perforin knockout mice are mediated by APO-1L. To define whether APO-1L is expressed on the surface o
Publikováno v:
European journal of immunology. 24(12)
APO-1/Fas (CD95) is a transmembrane receptor that transduces apoptotic signals within various cells including T and B cells. The APO-1 gene was found to be defective in lpr mice. In these mice insertion of a retrotransposon gives rise to transcriptio