Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Bernardo Moreira Soares Oliveira"'
Autor:
Vahid M. Harandi, Bernardo Moreira Soares Oliveira, Valérie Allamand, Ariana Friberg, Cibely C. Fontes-Oliveira, Madeleine Durbeej
Publikováno v:
Antioxidants, Vol 9, Iss 3, p 244 (2020)
Congenital muscular dystrophy with laminin α2 chain-deficiency (LAMA2-CMD) is a severe neuromuscular disorder without a cure. Using transcriptome and proteome profiling as well as functional assays, we previously demonstrated significant metabolic i
Externí odkaz:
https://doaj.org/article/90a1ec39ad814e69b3fac1dbb915b07e
Publikováno v:
PLoS ONE, Vol 12, Iss 8, p e0181950 (2017)
MicroRNAs (miRNAs) are short non-coding RNAs that modulate gene expression post-transcriptionally. Current evidence suggests that miR-21 plays a significant role in the progression of fibrosis in muscle diseases. Laminin-deficient congenital muscular
Externí odkaz:
https://doaj.org/article/b28700a4a284482fa3bf832d678ff389
Publikováno v:
PLoS Currents
Circulating microRNAs (miRNAs) are being considered as non-invasive biomarkers for disease progression and clinical trials. Congenital muscular dystrophy with deficiency of laminin α2 chain (LAMA2-CMD) is a very severe form of muscular dystrophy, fo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::5bf04aaa5f8fdf51163f4ecd24ebb8d5
https://pubmed.ncbi.nlm.nih.gov/30430039
https://pubmed.ncbi.nlm.nih.gov/30430039
Autor:
Zandra Körner, Madeleine Durbeej, Bernardo Moreira Soares Oliveira, Cibely C. Fontes-Oliveira, Vahid M. Harandi
Publikováno v:
Scientific Reports
Scientific Reports, Vol 8, Iss 1, Pp 1-15 (2018)
Scientific Reports, Vol 8, Iss 1, Pp 1-15 (2018)
Congenital muscular dystrophy with laminin α2 chain-deficiency (LAMA2-CMD) is a severe muscle disorder with complex underlying pathogenesis. We have previously employed profiling techniques to elucidate molecular patterns and demonstrated significan
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::07ca2b21ab82da0cca7786c50eef8492
http://europepmc.org/articles/PMC6214987
http://europepmc.org/articles/PMC6214987
Publikováno v:
PLoS Currents.
Circulating microRNAs (miRNAs) are being considered as non-invasive biomarkers for disease progression and clinical trials. Congenital muscular dystrophy with deficiency of laminin α2 chain (LAMA2-CMD) is a very severe form of muscular dystrophy, fo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::dc0ab7e195dd33ea639ebbfb62deb01a
https://doi.org/10.1371/currents.md.d0c203c018bc024f2f4c9791ecb05f88
https://doi.org/10.1371/currents.md.d0c203c018bc024f2f4c9791ecb05f88
Autor:
Bernardo Moreira Soares Oliveira, Kinga I. Gawlik, Johan Holmberg, Martina Svensson, Mikaela Einerborg, Madeleine Durbeej, Tomas Deierborg
Publikováno v:
Scientific Reports
A large number of human diseases are caused by chronic tissue injury with fibrosis potentially leading to organ failure. There is a need for more effective anti-fibrotic therapies. Congenital muscular dystrophy type 1A (MDC1A) is a devastating form o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::666dc569eab0840631b3cf6a89fbc564
http://europepmc.org/articles/PMC5345027
http://europepmc.org/articles/PMC5345027
Publikováno v:
PLoS ONE, Vol 12, Iss 8, p e0181950 (2017)
PLoS ONE
PLoS ONE
MicroRNAs (miRNAs) are short non-coding RNAs that modulate gene expression post-transcriptionally. Current evidence suggests that miR-21 plays a significant role in the progression of fibrosis in muscle diseases. Laminin-deficient congenital muscular
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::63770125de831ff614aeea2a91c4437d
http://europepmc.org/articles/PMC5542641?pdf=render
http://europepmc.org/articles/PMC5542641?pdf=render
Autor:
Bernardo Moreira Soares Oliveira, Heikki Kainulainen, Olli Ritvos, Mika Silvennoinen, Hongqiang Ma, Willem M.H. Hoogaars, Arja Pasternack, Juha J. Hulmi, Philippe Pierre
Publikováno v:
American Journal of Physiology-Endocrinology and Metabolism. 304:E41-E50
Loss of muscle mass and function occurs in various diseases. Myostatin blocking can attenuate muscle loss, but downstream signaling is not well known. Therefore, to elucidate associated signaling pathways, we used the soluble activin receptor IIb (sA
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4ce5e9782eb6d78fd7b9ac6d318ff009
https://doi.org/10.1152/ajpendo.00389.2012
https://doi.org/10.1152/ajpendo.00389.2012
Autor:
Willem M.H. Hoogaars, Olli Ritvos, Mika Silvennoinen, Bernardo Moreira Soares Oliveira, Heikki Kainulainen, Arja Pasternack, Juha J. Hulmi
Publikováno v:
American Journal of Physiology-Endocrinology and Metabolism. 305(2):E171-E182
The importance of adequate levels of muscle size and function and physical activity is widely recognized. Myostatin/activin blocking increases skeletal muscle mass but may decrease muscle oxidative capacity and can thus be hypothesized to affect volu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::02bce2e158c88c6d074df73842759ef4
https://doi.org/10.1152/ajpendo.00065.2013
https://doi.org/10.1152/ajpendo.00065.2013
Autor:
Bernardo Moreira Soares Oliveira, Reija Autio, Heikki Kainulainen, Mustafa Atalay, Juha J. Hulmi, Jaakko Hentilä, Konstantinos G. Papaioannou, Urho M. Kujala, Olli Ritvos, Ayhan Korkmaz, Keith C. DeRuisseau
Protein homeostasis in cells, proteostasis, is maintained through several integrated processes and pathways and its dysregulation may mediate pathology in many diseases including Duchenne muscular dystrophy (DMD). Oxidative stress, heat shock protein
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::673ceab3a05ee09e487f0818e829864f
http://hdl.handle.net/10138/229645
http://hdl.handle.net/10138/229645
