Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Bernard Landwehrmeyer"'
Autor:
Tiago A. Mestre, Cristina Sampaio, Wim Vandenberghe, Klaus Seppi, Erin Furr-Stimming, Anne Elizabeth Rosser, Melissa J. Nirenberg, Joaquim J. Ferreira, Anna Rita Bentivoglio, Filipe B. Rodrigues, Sarah J. Tabrizi, Gonçalo S Duarte, João Costa, Jan Roth, Jaime Kulisevsky, Francis O. Walker, Francisco Cardoso, Jean-Marc Burgunder, Jarosław Sławek, Anne-Catherine Bachoud-Lévi, Daniel O. Claassen, G Bernard Landwehrmeyer
Publikováno v:
Repositório Científico de Acesso Aberto de Portugal
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
© 2021 International Parkinson and Movement Disorder Society
Background: Huntington's disease (HD) is a rare neurodegenerative disorder with protean clinical manifestations. Its management is challenging, consisting mainly of off-label treatmen
Background: Huntington's disease (HD) is a rare neurodegenerative disorder with protean clinical manifestations. Its management is challenging, consisting mainly of off-label treatmen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bafc11b9f83da3287793893b776185f5
https://doi.org/10.1002/mds.28855
https://doi.org/10.1002/mds.28855
Autor:
Michael Trippel, Wolfgang Weber, Talib N. Omer, Simone Krebs, Bernard Landwehrmeyer, Guido Nikkhah, Thomas Prokop
Publikováno v:
Clinical and Experimental Neuroimmunology. 2:25-32
Objectives: Striatal transplantation of human fetal neurones as a therapeutic approach to treat Huntington’s disease is under investigation. The allogeneic graft material raises questions of immune response and immunosuppressive treatment. No data
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e28704c59395c5c75da1952feb8b1819
https://doi.org/10.1111/j.1759-1961.2011.00018.x
https://doi.org/10.1111/j.1759-1961.2011.00018.x
Autor:
Shane A. Worthge, Adeela Syed, Letizia Magnoni, Michela Camarri, Liliana B. Menalled, J. Lawrence Marsh, Andrea Caricasole, Marco Gianfriddo, Enrica Diodato, Tamas Lukacsovich, Ruth Luthi-Carter, Ozgun Gokce, Judy Purcell, Marianne R. Smith, Luisa Massai, Sylvie Ramboz, Stephen R. Wei, Davide Franceschini, Russell J. Thomas, Giuseppe Pollio, G Westerberg, Bernard Landwehrmeyer, Carla Scali, Brett A. Barbaro, Carol Murphy, Sarah J. Tabrizi
Publikováno v:
Human molecular genetics, vol 23, iss 11
Smith, MR; Syed, A; Lukacsovich, T; Purcell, J; Barbaro, BA; Worthge, SA; et al.(2014). A potent and selective sirtuin 1 inhibitor alleviates pathology in multiple animal and cell models of huntington's disease. Human Molecular Genetics, 23(11), 2995-3007. doi: 10.1093/hmg/ddu010. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/91m8n212
Smith, MR; Syed, A; Lukacsovich, T; Purcell, J; Barbaro, BA; Worthge, SA; et al.(2014). A potent and selective sirtuin 1 inhibitor alleviates pathology in multiple animal and cell models of huntington's disease. Human Molecular Genetics, 23(11), 2995-3007. doi: 10.1093/hmg/ddu010. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/91m8n212
Protein acetylation, which is central to transcriptional control as well as other cellular processes, is disrupted in Huntington's disease (HD). Treatments that restore global acetylation levels, such as inhibiting histone deacetylases (HDACs), are e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a945f412d35a8fd0b971b3108edc54b4
https://escholarship.org/uc/item/91m8n212
https://escholarship.org/uc/item/91m8n212
