Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Benjamin T. Noble"'
Autor:
Benjamin T. Noble, Faith H. Brennan, Yan Wang, Zhen Guan, Xiaokui Mo, Jan M. Schwab, Phillip G. Popovich
Publikováno v:
J Neurosci
Traumatic spinal cord injury (SCI) above the major spinal sympathetic outflow (T6 level) disinhibits sympathetic neurons from supraspinal control, causing systems-wide “dysautonomia.” We recently showed that remarkable structural remodeling and p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::efbf998e2fae75a610e6fbe87d71835a
https://europepmc.org/articles/PMC9053847/
https://europepmc.org/articles/PMC9053847/
Publikováno v:
Journal of Neuroimmunology. 321:1-11
Traumatic spinal cord injury (SCI) causes widespread damage to neurons, glia and endothelia located throughout the spinal parenchyma. In response to the injury, resident and blood-derived leukocytes orchestrate an intraspinal inflammatory response th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0d05d33ecbff2ad2a8edb6d1eea7d164
https://doi.org/10.1016/j.jneuroim.2018.05.007
https://doi.org/10.1016/j.jneuroim.2018.05.007
Autor:
Clay Harris, Faith H. Brennan, Xiaokui Mo, Cagla Eroglu, Hayes Davis, Yan Wang, Zhen Guan, Benjamin T. Noble, Phillip G. Popovich
Publikováno v:
SSRN Electronic Journal.
Spinal cord injury (SCI) damages supraspinal control over spinal autonomic circuitry. As a result, normally innocuous visceral or somatic stimuli (e.g. distended bladder or bowel) trigger pathological dysautonomia caused by uncontrolled reflex activa
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::aa8a796ffcdc40d027eea2ccc81530c0
https://doi.org/10.2139/ssrn.3470401
https://doi.org/10.2139/ssrn.3470401
Autor:
Hayes Davis, Benjamin T. Noble, Yan Wang, Xiaokui Mo, Faith H. Brennan, Cagla Eroglu, Zhen Guan, Clay Harris, Phillip G. Popovich, Adam R. Ferguson
Publikováno v:
Cell reports, vol 34, iss 4
Cell reports
Cell reports
SUMMARY After spinal cord injury (SCI), normally innocuous visceral or somatic stimuli can trigger uncontrolled reflex activation of sympathetic circuitry, causing pathological dysautonomia. We show that remarkable structural remodeling and plasticit
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5786441dc7676136dc6722edb82048e1
https://doi.org/10.1016/j.celrep.2020.108667
https://doi.org/10.1016/j.celrep.2020.108667