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pro vyhledávání: '"Benjamin S. Summers"'
Autor:
Yilan Zhen, Carlie L. Cullen, Raphael Ricci, Benjamin S. Summers, Sakina Rehman, Zubair M. Ahmed, Antoinette Y. Foster, Ben Emery, Robert Gasperini, Kaylene M. Young
Publikováno v:
Communications Biology, Vol 5, Iss 1, Pp 1-21 (2022)
Protocadherin 15 promotes lamellipodial and filopodial dynamics in oligodendrocyte progenitor cells by regulating Cdc42-Arp2/3 activity, but also suppresses ERK1/2 phosphorylation to reduce proliferation.
Externí odkaz:
https://doaj.org/article/5c6fb3dbd66a477395c3c0828c084902
Autor:
Kimberley A Pitman, Kaylene M. Young, Benjamin S. Summers, Solène Ferreira, Carlie L. Cullen, Shiwei Wang
Publikováno v:
The European Journal of Neuroscience
Myelin and axon losses are associated with cognitive decline in healthy ageing but are worse in people diagnosed with tauopathy. To determine whether tauopathy is also associated with enhanced myelin plasticity, we evaluated the behaviour of OPCs in
Autor:
Yilan Zhen, Carlie L. Cullen, Raphael Ricci, Benjamin S. Summers, Sakina Rehman, Zubair M. Ahmed, Antoinette Y. Foster, Ben Emery, Robert Gasperini, Kaylene M. Young
Publikováno v:
Communications biology. 5(1)
Oligodendrocyte progenitor cells (OPCs) express protocadherin 15 (Pcdh15), a member of the cadherin superfamily of transmembrane proteins. Little is known about the function of Pcdh15 in the central nervous system (CNS), however, Pcdh15 expression ca
Autor:
null Solène Ferreira, null Kimberley A Pitman, null Benjamin S Summers, null Shiwei Wang, null Kaylene M Young, null Carlie L Cullen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::9ff29e5792ca4e6ec8a841d1dbecb6c3
https://doi.org/10.1111/ejn.14726/v3/response1
https://doi.org/10.1111/ejn.14726/v3/response1
Autor:
Shiwei Wang, Kimberley A Pitman, Benjamin S. Summers, Carlie L. Cullen, Nicole Bye, Kaylene M. Young, Solène Ferreira
Publikováno v:
Journal of Neuroscience Research
In Alzheimer's disease, amyloid plaque formation is associated with the focal death of oligodendrocytes and soluble amyloid β impairs the survival of oligodendrocytes in vitro. However, the response of oligodendrocyte progenitor cells (OPCs) to earl