Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Benjamin R, Gerber"'
Autor:
Benjamin R. Gerber, Jeanne M. Nerbonne, Marie A. Kozel, David M. Ornitz, Angelika Lampert, Fernanda Laezza, Stephen G. Waxman, Sulayman D. Dib-Hajj, Anthony M. Rush
Publikováno v:
Molecular and Cellular Neuroscience. 42:90-101
The Intracellular Fibroblast Growth Factor (iFGF) subfamily includes four members (FGFs 11-14) of the structurally related FGF superfamily. Previous studies showed that the iFGFs interact directly with the pore-forming (alpha) subunits of voltage-gat
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::73aea8334bf3f19413a2ce8db78f462a
https://doi.org/10.1016/j.mcn.2009.05.007
https://doi.org/10.1016/j.mcn.2009.05.007
Publikováno v:
The Journal of Physiology. 586:1565-1579
Considerable experimental evidence has accumulated demonstrating a role for voltage-gated K+ (Kv) channel pore-forming (α) subunits of the Kv4 subfamily in the generation of fast transient outward K+, IA, channels. Immunohistochemical data suggest t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d73f4b9d8d390a8f54bd573d8cb6d164
https://doi.org/10.1113/jphysiol.2007.146597
https://doi.org/10.1113/jphysiol.2007.146597
Autor:
Jeanne M. Nerbonne, David M. Ornitz, Ann Marie Craig, Fernanda Laezza, Jun Yang Lou, Kathryn A. Yamada, Benjamin R. Gerber, Maolei Xiao, Hali Hartmann
Publikováno v:
The Journal of Physiology. 569:179-193
Genetic ablation of the fibroblast growth factor (Fgf) 14 gene in mice or a missense mutation in Fgf14 in humans causes ataxia and cognitive deficits. These phenotypes suggest that the neuronally expressed Fgf14 gene is essential for regulating norma
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3d64d74a3c6d2fc9d58d66058c198806
https://doi.org/10.1113/jphysiol.2005.097220
https://doi.org/10.1113/jphysiol.2005.097220
Publikováno v:
Journal of neurophysiology. 107(6)
Familial hemiplegic migraine type 1 (FHM-1), a rare hereditary form of migraine with aura and hemiparesis, serves as a good model for exploring migraine pathophysiology. The FHM-1 gene encodes the pore-forming CaV2.1 subunit of human P/Q-type voltage
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b87e4f88d088f06a5ad4d56491b60e84
https://pubmed.ncbi.nlm.nih.gov/22190617
https://pubmed.ncbi.nlm.nih.gov/22190617
Considerable experimental evidence has accumulated demonstrating a role for voltage-gated K(+) (Kv) channel pore-forming (alpha) subunits of the Kv4 subfamily in the generation of fast transient outward K(+), I(A), channels. Immunohistochemical data
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::1c7edb30cdce82a2695f5a1d0793caf9
https://europepmc.org/articles/PMC2375705/
https://europepmc.org/articles/PMC2375705/
Autor:
Fernanda Laezza, Jun Yang Lou, Marie A. Kozel, Benjamin R. Gerber, Jeanne M. Nerbonne, Hali Hartman, Ann Marie Craig, David M. Ornitz
Fibroblast growth factor 14 (FGF14) belongs to the intracellular FGF homologous factor subfamily of FGF proteins (iFGFs) that are not secreted and do not activate tyrosine kinase receptors. The iFGFs, however, have been shown to interact with the por
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6075c2351adf0fd326deaf93d127ff56
https://europepmc.org/articles/PMC6673376/
https://europepmc.org/articles/PMC6673376/
Autor:
Jun-Yang, Lou, Fernanda, Laezza, Benjamin R, Gerber, Maolei, Xiao, Kathryn A, Yamada, Hali, Hartmann, Ann Marie, Craig, Jeanne M, Nerbonne, David M, Ornitz
Publikováno v:
The Journal of physiology. 569(Pt 1)
Genetic ablation of the fibroblast growth factor (Fgf) 14 gene in mice or a missense mutation in Fgf14 in humans causes ataxia and cognitive deficits. These phenotypes suggest that the neuronally expressed Fgf14 gene is essential for regulating norma
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::64f46d05d83e4985ffceb901916e5103
https://pubmed.ncbi.nlm.nih.gov/16166153
https://pubmed.ncbi.nlm.nih.gov/16166153