Zobrazeno 1 - 10
of 27
pro vyhledávání: '"Benjamin M. Matta"'
Autor:
Jeremy M. Lott, Brian R. Rosborough, Bruce R. Blazar, Benjamin M. Matta, Lisa R. Mathews, Quan Liu, Heth R. Turnquist
Publikováno v:
The Journal of Immunology. 193:4010-4020
IL-33 is a recently characterized IL-1 family member that is proposed to function as an alarmin, or endogenous signal of cellular damage, as well as act as a pleiotropic cytokine. The ability of IL-33 to potentiate both Th1 and Th2 immunity supports
Autor:
Mark A. Ross, Angus W. Thomson, Benjamin M. Matta, Osamu Yoshida, Shoko Kimura, Shinichiro Yokota, David A. Geller, Lei Dou
Publikováno v:
American Journal of Transplantation. 14:1791-1805
Liver interstitial dendritic cells (DC) have been implicated in immune regulation and tolerance induction. We found that the transmembrane immuno-adaptor DNAX-activating protein of 12 kDa (DAP12) negatively regulated conventional liver myeloid (m) DC
Cell damage and death releases “alarmins”, or self-derived immunomodulatory molecules, that recruit and activate the immune system. Unfortunately, numerous processes critical to the transplantation of allogeneic materials result in the destructio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::85e5df26ca68cfdfe7d29f1ed365bc89
https://europepmc.org/articles/PMC5124552/
https://europepmc.org/articles/PMC5124552/
Autor:
Benjamin M. Matta, Heth R. Turnquist
Publikováno v:
Methods in Molecular Biology ISBN: 9781493931385
Thymic-derived, regulatory T cells (Treg) represent a subset of CD4(+) T cells that are required for normal immune homeostasis and suppression of unwanted responses against self-antigens (Ags) that prevent autoimmunity. Their role as immune regulator
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8fc017f4d8448645ddd0616a0977f8cd
https://doi.org/10.1007/978-1-4939-3139-2_3
https://doi.org/10.1007/978-1-4939-3139-2_3
Autor:
Chandrashekhar R. Gandhi, Angus W. Thomson, Donna B. Stolz, Tina L. Sumpter, Yoram Vodovotz, Benjamin M. Matta, Chao Huang, Anil Dangi
Publikováno v:
The Journal of Immunology. 189:3848-3858
Hepatic stellate cells (HSCs) are critical for hepatic wound repair and tissue remodeling. They also produce cytokines and chemokines that may contribute to the maintenance of hepatic immune homeostasis and the inherent tolerogenicity of the liver. T
Publikováno v:
The Journal of Immunology. 188:5227-5237
Plasmacytoid dendritic cells (pDCs) are highly specialized APCs that, in addition to their well-recognized role in anti-viral immunity, also regulate immune responses. Liver-resident pDCs are considerably less immunostimulatory than those from second
Autor:
Petar J. Popovic, Juan B. Ochoa, Benjamin M. Matta, Mazen S. Zenati, John P. Pribis, Jodie Bryk, Veronica Munera, David Caba
Publikováno v:
Annals of Surgery. 251:120-126
Objective To delineate the role of T-helper 2 (Th2) cytokines in the induction of trauma induced myeloid suppressor cells (TIMSC) and the regulation of nitric oxide production. Background Trauma induces myeloid cells that express CD11b+/Gr1+ and argi
Autor:
Benjamin M. Matta, Tina L. Sumpter, Yoram Vodovotz, Zhiliang Wang, Giorgio Raimondi, Natasha Corbitt, Angus W. Thomson, Mahesh Pillai
Publikováno v:
The Journal of Immunology. 184:624-636
Minimization of immunosuppression and donor-specific tolerance to MHC-mismatched organ grafts are important clinical goals. The therapeutic potential of regulatory T cells (Tregs) has been demonstrated, but conditions for optimizing their in vivo fun
Autor:
Benjamin M, Matta, Hēth R, Turnquist
Publikováno v:
Methods in molecular biology (Clifton, N.J.). 1371
Thymic-derived, regulatory T cells (Treg) represent a subset of CD4(+) T cells that are required for normal immune homeostasis and suppression of unwanted responses against self-antigens (Ags) that prevent autoimmunity. Their role as immune regulator
Publikováno v:
The Journal of Immunology. 176:2085-2094
T cell dysfunction that occurs after surgery or trauma is associated with a poor clinical outcome. We describe that myeloid suppressor cells expressing CD11b+/Gr-1+ markers invade the spleen after traumatic stress and suppress T cell function through