Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Benjamin J. Forred"'
Autor:
Benjamin J Forred, Darwin R Daugaard, Brianna K Titus, Ryan R Wood, Miranda J Floen, Michelle L Booze, Peter F Vitiello
Publikováno v:
PLoS ONE, Vol 12, Iss 1, p e0168777 (2017)
Mitochondria play a fundamental role in the regulation of cell death during accumulation of oxidants. High concentrations of atmospheric oxygen (hyperoxia), used clinically to treat tissue hypoxia in premature newborns, is known to elicit oxidative s
Externí odkaz:
https://doaj.org/article/bd80729894114bcfa6d082b27d3c1290
Autor:
Michelle L Baack, Benjamin J Forred, Tricia D Larsen, Danielle N Jensen, Angela L Wachal, Muhammad Ali Khan, Peter F Vitiello
Publikováno v:
PLoS ONE, Vol 11, Iss 8, p e0160818 (2016)
RATIONALE:Infants born to diabetic or obese mothers are at risk of respiratory distress and persistent pulmonary hypertension of the newborn (PPHN), conceivably through fuel-mediated pathogenic mechanisms. Prior research and preventative measures foc
Externí odkaz:
https://doaj.org/article/cf0134c95c844fb9b2c600320cce2cbc
Autor:
Y. Cao, Peter F. Vitiello, C. Esslinger, J. Hoffman, Benjamin J. Forred, Trent E. Tipple, Cassandra Aegerter, K. Bruening
Publikováno v:
D27. CELLULAR RESPONSES INFORMING LUNG REPAIR.
Autor:
Cody J. Lensing, Peter F. Vitiello, Benjamin J. Forred, Michael W. Amolins, Ruthellen H. Anderson, Cassandra Aegerter, Jared R. Mays
Publikováno v:
ChemMedChem. 13(16)
Consumption of Brassica vegetables provides beneficial effects due to organic isothiocyanates (ITCs), a resultant product of the enzymatic hydrolysis of glucosinolate secondary metabolites. The ITC L-sulforaphane (L-SFN) is the principle agent in bro
Publikováno v:
Free Radical Biology and Medicine. 75:167-177
The most common form of newborn chronic lung disease, bronchopulmonary dysplasia (BPD), is thought to be caused by oxidative disruption of lung morphogenesis which results in decreased pulmonary vasculature and alveolar simplification. Although cellu
Autor:
Muhammad Ali Khan, Peter F. Vitiello, Angela L. Wachal, Michelle L. Baack, Benjamin J. Forred, Danielle N. Jensen, Tricia D. Larsen
Publikováno v:
PLoS ONE
PLoS ONE, Vol 11, Iss 8, p e0160818 (2016)
PLoS ONE, Vol 11, Iss 8, p e0160818 (2016)
Rationale Infants born to diabetic or obese mothers are at risk of respiratory distress and persistent pulmonary hypertension of the newborn (PPHN), conceivably through fuel-mediated pathogenic mechanisms. Prior research and preventative measures foc
Autor:
Peter F. Vitiello, Michael W. Amolins, Kyle J. Roux, Benjamin J. Forred, Skyla Neuharth, Dae In Kim, Khatereh Motamedchaboki
Publikováno v:
Oxidative Medicine and Cellular Longevity, Vol 2016 (2016)
Oxidative Medicine and Cellular Longevity
Oxidative Medicine and Cellular Longevity
Thioredoxin-interacting protein (Txnip) acts as a negative regulator of thioredoxin function and is a critical modulator of several diseases including, but not limited to, diabetes, ischemia-reperfusion cardiac injury, and carcinogenesis. Therefore,
Autor:
Trent E. Tipple, J. Hoffman, Peter F. Vitiello, Benjamin J. Forred, Beth Mordhorst, A. Fairchild, Cassandra Aegerter
Publikováno v:
Free Radical Biology and Medicine. 128:S115
Bronchopulmonary Dysplasia (BPD) is a chronic lung disease that affects pre-term infants whose lungs are unable to support life outside of the womb. These babies receive therapeutic oxygen which compensates for poor lung function but also causes oxid
Autor:
Benjamin J. Forred, Cassandra Aegerter, Peter F. Vitiello, Xaviera Thomas, Alexandra Fairchild, Erika Hernandez
Publikováno v:
Free Radical Biology and Medicine. 112:182
Thioredoxin 1 (Trx1) regulates redox signaling through reduction of cysteine thiols in protein substrates. Impaired Trx1 function has been implicated in the pathogenesis of several neurodegenerative, pulmonary, immune, metabolic and cardiovascular di
Publikováno v:
The FASEB Journal. 28
Many cell signaling processes are dependent on redox-dependent interactions through the thioredoxin (Trx) family. Thioredoxins regulate the reduction-oxidation state of protein substrates through antioxidant enzymatic activities. Thioredoxins have a