Zobrazeno 1 - 8 of 8 pro vyhledávání: '"Benjamin F. Wyse"'
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Akademický článek
Renal denervation does not affect hypertension or the renin-angiotensin system in a rodent model of juvenile-onset polycystic kidney disease: clinical implications
Autor: Sheran Li, Cara M. Hildreth, Ahmed A. Rahman, Sean A. Barton, Benjamin F. Wyse, Chai K. Lim, Paul M. Pilowsky, Jacqueline K. Phillips
Publikováno v: Scientific Reports, Vol 11, Iss 1, Pp 1-16 (2021)
Abstract We examined the effect of total and afferent renal denervation (RDN) on hypertension and the renin-angiotensin system (RAS) in a rodent model of juvenile-onset polycystic kidney disease (PKD). Lewis Polycystic Kidney (LPK) and control rats r
Externí odkaz: https://doaj.org/article/48a77c43345347dcab9d38e3bb3a6bbb
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2
Renal denervation does not affect hypertension or the renin-angiotensin system in a rodent model of juvenile-onset polycystic kidney disease: clinical implications
Autor: Chai K. Lim, Ahmed A. Rahman, Jacqueline K. Phillips, Cara M. Hildreth, Paul M. Pilowsky, Sean A. Barton, Sheran Li, Benjamin F. Wyse
Publikováno v: Scientific Reports
Scientific Reports, Vol 11, Iss 1, Pp 1-16 (2021)
We examined the effect of total and afferent renal denervation (RDN) on hypertension and the renin-angiotensin system (RAS) in a rodent model of juvenile-onset polycystic kidney disease (PKD). Lewis Polycystic Kidney (LPK) and control rats received t
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a7ee85ea09f6bc5bc32fe92f35a777ec
http://europepmc.org/articles/PMC8275789
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3
Protective cardiorenal effects of spironolactone in a rodent model of polycystic kidney disease
Autor: Omar Z. Ameer, Benjamin F. Wyse, Conor F. Underwood, Thamarasee M. Jeewandara, Rochelle Boyd, Jacqueline K. Phillips
Publikováno v: Clinical and Experimental Pharmacology and Physiology. 42:353-360
Summary Studies were performed to examine the contribution of aldosterone to the pathogenesis of cardiovascular and renal disease in a rodent model of genetic kidney disease. Spironolactone (20 mg/kg per day) was administered in water to mixed sex Le
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::27d6f9115a4d0b66e3b2627dcb6be92b
https://doi.org/10.1111/1440-1681.12372
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5
Uraemia: an unrecognized driver of central neurohumoral dysfunction in chronic kidney disease?
Autor: Rochelle Boyd, Jacqueline K. Phillips, Cara M. Hildreth, Ann K. Goodchild, Benjamin F. Wyse, Conor F. Underwood
Publikováno v: Acta physiologica (Oxford, England). 219(1)
Chronic kidney disease (CKD) carries a large cardiovascular burden in part due to hypertension and neurohumoral dysfunction - manifesting as sympathetic overactivity, baroreflex dysfunction and chronically elevated circulating vasopressin. Alteration
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7fea1987a58b629778aab989657c7eb0
https://pubmed.ncbi.nlm.nih.gov/27247097
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6
Chronic treatment with tempol does not significantly ameliorate renal tissue hypoxia or disease progression in a rodent model of polycystic kidney disease
Autor: Priyadharshani Kalaignanasundaram, Sharon D. Ricardo, Benjamin F. Wyse, Amany Abdelkader, Paul K. Witting, Jacqueline K. Phillips, Alice Ding, Hyun Bo Kim, Roger G. Evans, Brad R. S Broughton
Publikováno v: Clinical and experimental pharmacologyphysiology. 39(11)
Summary In the present study, we tested whether polycystic kidney disease (PKD) is associated with renal tissue hypoxia and oxidative stress, which, in turn, contribute to the progression of cystic disease and hypertension. Lewis polycystic kidney (L
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::07ecd59d03cf875e27528dcbd8501d8c
https://pubmed.ncbi.nlm.nih.gov/23006058
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