Zobrazeno 1 - 10
of 42
pro vyhledávání: '"Benjamin, M.L."'
Autor:
Hilmers, Torben, Leroy, Benjamin M.L., Bae, Soyeon, Hahn, W. Andreas, Hochrein, Sophia, Jacobs, Martin, Lemme, Hannes, Müller, Jörg, Schmied, Gerhard, Weisser, Wolfgang W., Pretzsch, Hans
Publikováno v:
In Forest Ecology and Management 1 December 2023 549
Autor:
Drum, Benjamin M.L., Yuan, Can, Li, Lei, Liu, Qinghang, Wordeman, Linda, Santana, L. Fernando
Publikováno v:
In Journal of Molecular and Cellular Cardiology April 2016 93:32-43
Publikováno v:
In Journal of Molecular and Cellular Cardiology January 2014 66:63-71
Publikováno v:
Am J Physiol Cell Physiol
Excitation-contraction (EC) coupling is the coordinated process by which an action potential triggers cardiac myocyte contraction. EC coupling is initiated in dyads where the junctional sarcoplasmic reticulum (jSR) is in tight proximity to the sarcol
Autor:
Leroy, Benjamin M.L., Gossner, Martin M., Ferrini, Gabriele, Seibold, Sebastian, Lauer, Florian P.M., Petercord, Ralf, Eichel, Peter, Jaworek, Jessica, Weisser, Wolfgang W.
Publikováno v:
Environmental Toxicology and Chemistry, 40 (4)
Internal feeding is considered to shield sessile herbivorous insects from exposure to nonsystemic insecticides aerially sprayed against forest defoliators, although this has not been tested. It is, however, established that leaf damage caused by defo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::437da02c182de9431c453213a15e3420
https://hdl.handle.net/20.500.11850/477994
https://hdl.handle.net/20.500.11850/477994
Publikováno v:
In Computer-Aided Design 2005 37(8):799-814
Autor:
Haifeng Yin, Jing Li, L. Fernando Santana, Yi Chen, Stephen W. Luckey, Merle L. Gilbert, Xiaoyun Guo, Lei Li, G. Stanley McKnight, John D. Scott, Qinghang Liu, Benjamin M.L. Drum
Publikováno v:
Cardiovascular research, vol 113, iss 2
Li, L; Li, J; Drum, BM; Chen, Y; Yin, H; Guo, X; et al.(2017). Loss of AKAP150 promotes pathological remodelling and heart failure propensity by disrupting calcium cycling and contractile reserve. Cardiovascular Research, 113(2), 147-159. doi: 10.1093/cvr/cvw221. UC Davis: Retrieved from: http://www.escholarship.org/uc/item/0912z16q
Li, L; Li, J; Drum, BM; Chen, Y; Yin, H; Guo, X; et al.(2017). Loss of AKAP150 promotes pathological remodelling and heart failure propensity by disrupting calcium cycling and contractile reserve. Cardiovascular Research, 113(2), 147-159. doi: 10.1093/cvr/cvw221. UC Davis: Retrieved from: http://www.escholarship.org/uc/item/0912z16q
© 2016 The Author. Aims Impaired Ca2+cycling and myocyte contractility are a hallmark of heart failure triggered by pathological stress such as hemodynamic overload. The A-Kinase anchoring protein AKAP150 has been shown to coordinate key aspects of
Publikováno v:
Journal of Molecular and Cellular Cardiology. 66:63-71
Ca(2+) flux through l-type CaV1.2 channels shapes the waveform of the ventricular action potential (AP) and is essential for excitation-contraction (EC) coupling. Timothy syndrome (TS) is a disease caused by a gain-of-function mutation in the CaV1.2
Publikováno v:
Journal of Molecular and Cellular Cardiology. 81:15-17
The junctional sarcoplasmic reticulum (jSR) is an important and unique ER subdomain in the adult myocyte that concentrates resident proteins to regulate Ca2+ release. To investigate cellular mechanisms for sorting and trafficking proteins to jSR, we
Autor:
Merle L. Gilbert, Haifeng Yin, Benjamin M.L. Drum, Lei Li, Jing Li, G S McKnight, Xiaoyun Guo, Stephen W. Luckey, Yi Chen, Luis Fernando Santana, John D. Scott, Qinghang Liu
Publikováno v:
Circulation Research. 119
Impaired Ca 2+ cycling and myocyte contractility are a hallmark of heart failure triggered by pathological stress such as hemodynamic overload. The A-Kinase anchoring protein AKAP150 has been shown to coordinate key aspects of adrenergic regulation o