Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Benedikt Beckert"'
Autor:
Anna Zakrzewicz, Celina Würth, Benedikt Beckert, Simon Feldhoff, Katrien Vanderheyden, Stian Foss, Jan Terje Andersen, Hans de Haard, Peter Verheesen, Vladimir Bobkov, Ritva Tikkanen
Publikováno v:
Cells, Vol 11, Iss 10, p 1700 (2022)
The authors wish to make the following changes to their paper [...]
Externí odkaz:
https://doaj.org/article/61896ac700a94631a0d52eeb793152df
Autor:
Anna Zakrzewicz, Celina Würth, Benedikt Beckert, Simon Feldhoff, Katrien Vanderheyden, Stian Foss, Jan Terje Andersen, Hans de Haard, Peter Verheesen, Vladimir Bobkov, Ritva Tikkanen
Publikováno v:
Cells, Vol 11, Iss 6, p 942 (2022)
Pemphigus vulgaris is an autoimmune blistering disease of the epidermis, caused by autoantibodies against desmosomal proteins, mainly desmogleins 1 and 3, which induce an impairment of desmosomal adhesion and blister formation. Recent findings have s
Externí odkaz:
https://doaj.org/article/94bff078217b4747b791e79e694256c3
Autor:
Jens Weingarten, Melanie Laßek, Benjamin F. Mueller, Marion Rohmer, Dominic Baeumlisberger, Benedikt Beckert, Jens Ade, Patricia Gogesch, Amparo Acker-Palmer, Michael Karas, Walter Volknandt
Publikováno v:
Proteomes, Vol 3, Iss 2, Pp 74-88 (2015)
Neurotransmitter release as well as structural and functional dynamics at the presynaptic active zone (PAZ) comprising synaptic vesicles attached to the presynaptic plasma membrane are mediated and controlled by its proteinaceous components. Here we
Externí odkaz:
https://doaj.org/article/b8d1293d2bdc4cdf8fbe0dc83f95f3e0
Autor:
Stian Foss, Peter Verheesen, Celina Würth, Ritva Tikkanen, Simon Feldhoff, Vladimir Bobkov, Anna Zakrzewicz, Hans Dehaard, Jan Terje Andersen, Benedikt Beckert, Katrien Vanderheyden
Publikováno v:
Cells; Volume 11; Issue 6; Pages: 942
Pemphigus vulgaris is an autoimmune blistering disease of the epidermis, caused by autoantibodies against desmosomal proteins, mainly desmogleins 1 and 3, which induce an impairment of desmosomal adhesion and blister formation. Recent findings have s
Autor:
Michael Hertl, Rüdiger Eming, Thomas Schmitt, Ritva Tikkanen, Roberto Maglie, Maria Llamazares-Prada, Christoph Hudemann, Takashi Hashimoto, Benedikt Beckert, Dario Didona, Jens Waschke
Publikováno v:
Journal of Investigative Dermatology. 142:915-923.e3
Pemphigus is a potentially lethal autoimmune bullous skin disorder, which is associated with IgG autoantibodies against desmoglein (DSG) 3 and DSG1. Notably, a subset of patients with pemphigus presents with a similar clinical phenotype in the absenc
Aβ42 oligomers impair the bioenergetic activity in hippocampal synaptosomes derived from APP-KO mice
Publikováno v:
Biological Chemistry
Employing hippocampal synaptosomes from amyloid precursor protein (APP)-deleted mice we analyzed the immediate effects of amyloid beta peptide 42 (Aβ42) peptide in its oligomeric or fibrillar assembly or of soluble amyloid precursor protein alpha (s
Autor:
Michael Hertl, P. Verheesen, K. Vanderheyden, S. Feldhoff, Ritva Tikkanen, V. Bobkov, Benedikt Beckert, I. Stoykov, Anna Zakrzewicz, C. Würth
Publikováno v:
Journal of Investigative Dermatology. 141:S154
Autor:
Francesca Panico, Benedikt Beckert, Ritva Tikkanen, Rüdiger Eming, Robert Pollmann, Antje Banning
Publikováno v:
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 20, Iss 13, p 3113 (2019)
Volume 20
Issue 13
International Journal of Molecular Sciences, Vol 20, Iss 13, p 3113 (2019)
Volume 20
Issue 13
Pemphigus Vulgaris is an autoimmune disease that results in blister formation in the epidermis and in mucosal tissues due to antibodies recognizing desmosomal cadherins, mainly desmoglein-3 and -1. Studies on the molecular mechanisms of Pemphigus hav
Autor:
Anton Petcherski, Aleksandra Somogyi, Ritva Tikkanen, Frances M. Platt, David A. Priestman, Mylene Huebecker, Benedikt Beckert, Susan L. Cotman, Antje Banning, Mika O. Ruonala
Publikováno v:
International Journal of Molecular Sciences; Volume 19; Issue 2; Pages: 625
International Journal of Molecular Sciences, Vol 19, Iss 2, p 625 (2018)
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 19, Iss 2, p 625 (2018)
International Journal of Molecular Sciences
Juvenile neuronal ceroid lipofuscinosis (JNCL) is caused by mutations in the CLN3 gene. Most JNCL patients exhibit a 1.02 kb genomic deletion removing exons 7 and 8 of this gene, which results in a truncated CLN3 protein carrying an aberrant C-termin