Zobrazeno 1 - 10 of 15 pro vyhledávání: '"Bendik Christian Brinchmann"'
1
Combustion Particle-Induced Changes in Calcium Homeostasis: A Contributing Factor to Vascular Disease?
Autor: Dominique Lagadic-Gossmann, Johan Øvrevik, Jørn A. Holme, Eric Le Ferrec, Bendik Christian Brinchmann
Publikováno v: Cardiovascular Toxicology
Cardiovascular Toxicology, 2019, 19 (3), pp.198-209. ⟨10.1007/s12012-019-09518-9⟩
Cardiovascular Toxicology, Humana Press, 2019, 19 (3), pp.198-209. ⟨10.1007/s12012-019-09518-9⟩
Air pollution is the leading environmental risk factor for disease and premature death in the world. This is mainly due to exposure to urban air particle matter (PM), in particular, fine and ultrafine combustion-derived particles (CDP) from traffic-r
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5bb1be2a1b9afab1c3f4d1afd1f31859
https://doi.org/10.1007/s12012-019-09518-9
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2
Organic chemicals from diesel exhaust particles affects intracellular calcium, inflammation and β-adrenoceptors in endothelial cells
Autor: Dominique Lagadic-Gossmann, Johan Øvrevik, Eric Le Ferrec, Bendik Christian Brinchmann, Normand Podechard, Jørn A. Holme
Publikováno v: Toxicology Letters
Toxicology Letters, Elsevier, 2019, 302, pp.18-27. ⟨10.1016/j.toxlet.2018.11.009⟩
Toxicology Letters, 2019, 302, pp.18-27. ⟨10.1016/j.toxlet.2018.11.009⟩
Exposure to diesel exhaust particles (DEP) may contribute to endothelial dysfunction and cardiovascular disease. DEP, extractable organic material from DEP (DEP-EOM) and certain PAHs seem to trigger [Ca2+]i increase as well as inflammation via GPCRs
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::891d9d27af65c709a35f09924ac39d3c
https://doi.org/10.1016/j.toxlet.2018.11.009
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6
Are the health risks of moist oral snuff (snus) underestimated?
Autor: Gunn Elisabeth, Vist, Tom Kristian, Grimsrud, Håkon, Valen, Rune, Becher, Bendik Christian, Brinchmann, Ida-Kristin Ørjasæter, Elvsaas, Jan, Alexander
Publikováno v: Tidsskrift for den Norske laegeforening : tidsskrift for praktisk medicin, ny raekke. 140(9)
The use of moist oral snuff (snus) has increased significantly, particularly among young adults who have not previously smoked. Snus increases the risk of cancer, cardiovascular disease, type-2 diabetes and birth defects.
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::2cf4a99ed8650404176088a0fc7c7f0c
https://pubmed.ncbi.nlm.nih.gov/32549010
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9
Pro-inflammatory effects of crystalline- and nano-sized non-crystalline silica particles in a 3D alveolar model
Autor: Johan Øvrevik, Tommaso Serchi, Jørn A. Holme, Magne Refsnes, Arno C. Gutleb, Tonje Skuland, Marit Låg, Bendik Christian Brinchmann
Publikováno v: 17:13
Particle and Fibre Toxicology
Particle and Fibre Toxicology, Vol 17, Iss 1, Pp 1-18 (2020)
Background Silica nanoparticles (SiNPs) are among the most widely manufactured and used nanoparticles. Concerns about potential health effects of SiNPs have therefore risen. Using a 3D tri-culture model of the alveolar lung barrier we examined effect
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fe35f86fd6457d89cfd9b3885e03dca9
https://hdl.handle.net/11250/2726876
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10
Lipophilic components of diesel exhaust particles induce pro-inflammatory responses in human endothelial cells through AhR dependent pathway(s)
Autor: Johan Øvrevik, Magne Refsnes, Dominique Lagadic-Gossmann, Jørn A. Holme, Eric Le Ferrec, Bendik Christian Brinchmann, Klara Kukowski, Alena Kubátová, Krisztina Szöke, Arno C. Gutleb, Jan E. Brinchmann, Tonje Skuland, Mia H. Rambøl, Marit Låg, Elisa Moschini, Per E. Schwarze
Publikováno v: Particle and Fibre Toxicology, Vol 15, Iss 1, Pp 1-17 (2018)
Particle and Fibre Toxicology
Particle and Fibre Toxicology, 2018, 15 (1), pp.21. ⟨10.1186/s12989-018-0257-1⟩
Particle and Fibre Toxicology, BioMed Central, 2018, 15 (1), pp.21. ⟨10.1186/s12989-018-0257-1⟩
Background Exposure to traffic-derived particulate matter (PM), such as diesel exhaust particles (DEP), is a leading environmental cause of cardiovascular disease (CVD), and may contribute to endothelial dysfunction and development of atherosclerosis
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6a3fbec240e80ef4cdbd1af45ec5ad10
http://link.springer.com/article/10.1186/s12989-018-0257-1
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