Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Ben Buller"'
Autor:
Tao Yan, Michael Chopp, Xinchun Ye, Zhongwu Liu, Alex Zacharek, Yisheng Cui, Cynthia Roberts, Ben Buller, Jieli Chen
Publikováno v:
Neurobiology of Disease, Vol 46, Iss 1, Pp 157-164 (2012)
Background and objective: We investigated axonal plasticity in the bilateral motor cortices and the long term therapeutic effect of Niaspan on axonal remodeling after stroke in type-1 diabetic (T1DM) rats. Experimental approaches: T1DM was induced in
Externí odkaz:
https://doaj.org/article/bef88a91f6ec445197eb26335623008b
Autor:
Oyinkansola Osobamiro, Xuguang Zheng, Michael Chopp, Ben Buller, Mark Katakowski, Thomas J. Rogers, Wayne Shu, Feng Jiang, Yong Lu
Publikováno v:
Cancer Letters. 335:201-204
Exosomes are 30–150 nm vesicles secreted by a wide range of mammalian cells that can contain microRNA (miRNA). To test if marrow stromal cell (MSC) exosomes could be used as a vehicle for delivery of anti-tumor miRNAs, we transfected MSCs with a mi
Autor:
Yi Zhang, Xinli Wang, Ben Buller, Hongqi Xin, Yi Li, Zheng Gang Zhang, Xia Shang, Michael Chopp, Mark Katakowski
Publikováno v:
Stem Cells. 30:1556-1564
Multipotent mesenchymal stromal cells (MSCs) have potential therapeutic benefit for the treatment of neurological diseases and injury. MSCs interact with and alter brain parenchymal cells by direct cell-cell communication and/or by indirect secretion
Autor:
Jieli Chen, Cynthia Roberts, Alex Zacharek, Ben Buller, Yisheng Cui, Zhongwu Liu, Michael Chopp, Xinchun Ye, Tao Yan
Publikováno v:
Neurobiology of Disease, Vol 46, Iss 1, Pp 157-164 (2012)
Background and objective: We investigated axonal plasticity in the bilateral motor cortices and the long term therapeutic effect of Niaspan on axonal remodeling after stroke in type-1 diabetic (T1DM) rats. Experimental approaches: T1DM was induced in
Autor:
Michael Chopp, Xinli Wang, Ann Hozeska-Solgot, Ben Buller, Rui Lan Zhang, Xianshuang Liu, Li Zhang, Zheng Gang Zhang
Publikováno v:
FEBS Journal. 277:4299-4307
MicroRNAs are small RNAs that attenuate protein expression by complementary binding to the 3'-UTR of a target mRNA. Currently, very little is known about microRNAs after cerebral ischemia. In particular, microRNA-21 (miR-21) is a strong antiapoptotic
Autor:
Mei Lu, Zheng Gang Zhang, Smita Savant-Bhonsale, Yi Li, Jade Borneman, Ben Buller, Michael Chopp, Stanton B. Elias, Jing Zhang
Publikováno v:
Journal of Cerebral Blood Flow & Metabolism. 29:1166-1174
Oligodendrocytes are sensitive to ischemic damage. The Sonic hedgehog (Shh) pathway is critical in oligodendrogenesis; Gli1 is the principal effector of Shh signaling. We investigated oligodendrogenesis and Shh/Gli1 pathway activation after bone marr
Autor:
Danping Zhao, Yanting Jiang, Dan Morris, James Jiang, Michael Chopp, Xianshuang Liu, Ben Buller, Zheng Gang Zhang, Li Zhang
Publikováno v:
Stroke. 41(5)
Background and Purpose— Treatment with a selective proteasome inhibitor, VELCADE, in combination with tissue plasminogen activator (tPA) extended the therapeutic window to 6 hours in young rats after stroke. However, stroke is a major cause of deat
Autor:
Sara C. Gregg, Michael Chopp, Ann Hozeska-Solgot, Zheng Gang Zhang, Rui Lan Zhang, Xian Shuang Liu, Mei Lu, Ben Buller
Ischemic stroke stimulates neurogenesis in the adult rodent brain. The molecules underlying stroke-induced neurogenesis have not been fully investigated. Using real-time reverse transcription-PCR, we found that stroke substantially up-regulated angio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::39ae80cac8053e92bc96a27870d3e766
https://europepmc.org/articles/PMC2755676/
https://europepmc.org/articles/PMC2755676/
Autor:
Zheng Gang Zhang, Ann Hozeska-Solgot, Xian Shuang Liu, Sara R Gregg, Ben Buller, Michael Chopp, Rui Lan Zhang, Xue Guo Zhang
Publikováno v:
Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism. 29(2)
Stroke increases neuroblasts in the subventricular zone (SVZ) of the lateral ventricle and these neuroblasts migrate toward the ischemic boundary to replace damaged neurons. Using brain slices from the nonischemic adult rat and transgenic mice that e