Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Bei-Bei Dai"'
Autor:
Xiao-Shan Wu, Bei-Bei Dai, Wei Hu, Chuanjun Zhu, Fei Zhu, Xuewen Qian, Ke Wang, Feihu Chen, Yong Chen, Ren-Peng Zhou, Sujing Song
Publikováno v:
Journal of Cellular and Molecular Medicine
Rheumatoid arthritis (RA) is a chronic, systemic autoimmune disease that eventually leads to joint deformities and loss of joint function. Previous studies have demonstrated a close relationship between autophagy and the development of RA. Although a
Autor:
Feihu Chen, Ya-Ya Xie, Sheng-Qin Zu, Xiao-Shan Wu, Chuanjun Zhu, Yu-Bin Feng, Ge Li, Jin-Fang Ge, Bei-Bei Dai, Ren-Peng Zhou, Yue Li, Zhi-Sen Wang
Publikováno v:
Cell biology internationalREFERENCES. 44(10)
The pyroptosis is a causative agent of rheumatoid arthritis, a systemic autoimmune disease merged with degenerative articular cartilage. Nevertheless, the precise mechanism of extracellular acidosis on chondrocyte pyroptosis is largely unclear. Acid-
Autor:
Ren-Peng Zhou, Ya-Ya Xie, Zhi-Qiang Wang, Wei-Jie Yang, Bei-Bei Dai, Jin-Fang Ge, Wen-Lin Ni, Zhi-Sen Wang, Fei-Hu Chen, Wei Hu, Xiao-Shan Wu
Publikováno v:
Gene. 642:230-240
Acid hydrarthrosis is another important pathological character in rheumatoid arthritis (RA), and acid-sensing ion channel 1a (ASIC1a) plays a destructive role in acidosis-induced articular chondrocyte cytotoxicity. Recently, ASIC2a has been reported
Autor:
Jin-Fang Ge, Ren-Peng Zhou, Xiao-Shan Wu, Fei-Hu Chen, Ya-Ya Xie, Yue Li, Zhi-Sen Wang, Sheng-Qin Zu, Bei-Bei Dai, Zhi-Qiang Wang
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1864:162-177
The acute-phase proinflammatory cytokines interleukin-1β (IL-1β) and tumor necrosis factor-α (TNF-α) demonstrate high-level expression and pleiotropic biological effects, and contribute to the progression and persistence of rheumatoid arthritis (
Effects of autophagy on acid-sensing ion channel 1a-mediated apoptosis in rat articular chondrocytes
Autor:
Ren-Peng Zhou, Bei-Bei Dai, Wei Hu, Yue Li, Xiao-Shan Wu, Yue-Jiao Qian, Fei-Hu Chen, Ya-Ya Xie, Jin-Fang Ge
Publikováno v:
Molecular and Cellular Biochemistry. 443:181-191
Rheumatoid arthritis (RA) is a degenerative joint disease that is caused by multiple pathogenic factors. However, the precise etiology of RA is still unknown. Our previous studies demonstrated that acid-sensing ion channel 1a (ASIC1a)-mediated articu
Autor:
Chao, Zhang, Ming-Hao, Shan, Dan-Ning, Xu, Ming-Zhe, Gu, Bei-Bei, Dai, Xuan, Ji, Jing-Mei, Sun
Publikováno v:
Huan jing ke xue= Huanjing kexue. 39(10)
The advanced treatment of treated petrochemical water by Ozone-Biological Activated Carbon (O
Autor:
Yong Chen, Feihu Chen, Ren-Peng Zhou, Jin-Fang Ge, Chuanjun Zhu, Zhi-Qiang Wang, Fei Zhu, Yu-Bin Feng, Bei-Bei Dai, Sujing Song
Publikováno v:
Biochemical and biophysical research communications. 504(4)
Necroptosis, a necrotic cell death pathway regulated by receptor interacting protein (RIP) 1 and 3, plays a key role in pathophysiological processes, including rheumatoid arthritis (RA). However, whether necroptosis is involved in RA articular cartil
Autor:
Jingfang Ge, Bei-Bei Dai, Ke Wang, Feihu Chen, Cong Wang, Sheng-Qin Zu, Yao Meng, Ren-Peng Zhou, Jinging Ruan, Ge Li, Yue Li, Yue-Jiao Qian
Publikováno v:
Gene. 683
4-amino-2-trifluoromethyl-phenyl retinate (ATPR), a novel all-trans retinoic acid (ATRA) derivative designed and synthesized by our team, has been demonstrated its anti-tumor effect through inducing differentiation and inhibiting proliferation. Eukar
Autor:
Zhi-Qiang Wang, Xiao-Shan Wu, Bei-Bei Dai, Feihu Chen, Jin-Fang Ge, Zhi-Sen Wang, Yue Li, Ren-Peng Zhou, Ya-Ya Xie, Sheng-Qin Zu
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1865:263-264
ASIC1a Promotes Acid-Induced Autophagy in Rat Articular Chondrocytes through the AMPK/FoxO3a Pathway
Autor:
Ya-Ya Xie, Sheng-Qin Zu, Xiao-Shan Wu, Yong Chen, Ren-Peng Zhou, Jin-Fang Ge, Bei-Bei Dai, Fei-Hu Chen, Fei Zhu, Ge Li, Zhi-Sen Wang
Publikováno v:
International Journal of Molecular Sciences; Volume 18; Issue 10; Pages: 2125
International Journal of Molecular Sciences, Vol 18, Iss 10, p 2125 (2017)
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 18, Iss 10, p 2125 (2017)
International Journal of Molecular Sciences
Acid-sensing ion channel 1a (ASIC1a) is a member of the extracellular H+-activated cation channels family. Our previous studies suggested that ASIC1a contributed to acid-induced rat articular chondrocytes autophagy. However, its potential mechanisms